缺氧的細胞 的英文怎麼說

中文拼音 [quēyǎngdexìbāo]
缺氧的細胞 英文
hypoxic cell
  • 缺 : Ⅰ動詞1 （缺乏; 短少） be short of; lack 2 （殘缺） be missing; be incomplete 3 （該到而未到） be ...
  • 氧 : 名詞[化學] （氣體元素） oxygen （o）
  • 的 : 4次方是 The fourth power of 2 is direction
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 缺氧 : anoxia; hypoxia; oxygen deficit; oxygen lack缺氧代謝 anaerobic metabolism; 缺氧環境 oxygen defici...
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  1. He found that irradiation of micrococcus and of murine lymphoma cells under anoxia gave fewer singlestrand breaks.

    他發現，在缺氧時照射小球菌和小鼠琳巴瘤細胞產生的單鏈斷裂較少。
  2. Effects of naloxone on rat cortical neuron apoptosis induced by anoxia

    納洛酮對體外培養的缺氧大鼠皮質神經元細胞凋亡的影響
  3. Cytoarchitectonics of neuronal nitric oxide synthase immunopositve neurons in caudate putame of rats with cerebral ischemia

    腦缺血大鼠尾殼核神經元型一氧化氮合酶免疫陽性神經元的細胞構築
  4. Protective effect of c - fos antisense gene recombinant transfection on ischemic hypoxic cardiomyocytes

    反義基因轉染對缺血缺氧心肌細胞的保護作用
  5. Protective effect of hyperoxia liquid on multiple organ damage in rabbit during ischemia reperfusion

    高氧液對缺血再灌注家兔多臟器細胞損傷的保護作用
  6. Hypoxia affects the osteoblasts proliferation and apoptosis

    缺氧對成骨細胞增殖及凋亡的影響
  7. Protective role of xingnao qizhi capsule in hypoxia injury of pc12 cell

    12細胞缺氧損傷的保護作用
  8. Response of proliferation of neural stem cells from rats ' embryos to hypoxia and the protective role of angelica sinensis

    鼠胚神經幹細胞增殖對缺氧的反應及當歸的保護作用
  9. Protective effect of brain - derived neurotrophic factor on hypoxic cultured cortical neurons and its cellular signal transduction pathway

    腦源性神經營養因子對缺氧神經元的保護作用及其細胞內信號傳遞
  10. Effect of cgrp on intracellular calcium concentration in cardiomyocytes under is chemia and hypoxia conditions

    降鈣素基因相關肽對心肌細胞缺血缺氧狀態下胞內鈣的影響
  11. Effects of ginkgo biloba extract 50 on hypoxia induced endothelial dysfunction

    50對缺氧致內皮細胞功能的影響
  12. The effects of simvastatin on expression of nitric oxide synthase in cultured human endothelial cells dur ing hypoxia - reoxygenation

    缺氧復氧損傷對血管內皮細胞一氧化氮合成酶基因表達的影響及辛伐他汀的干預作用
  13. Expression of intercellular adhesion molecule - 1 and activity of myeloperoxidase in diabetic rats after cerebral ischemiareperfusion

    糖尿病大鼠腦缺血再灌注后細胞間黏附分子1表達和髓過氧化物酶活性的變化
  14. Jin ke, yu liu, xing long et al. up - regulation of vascular endothelial growth factor in synovial fibroblasts from human temporomandibular joint by hypoxia. j oral pathol med 2007, 36 ( 5 ) : 290 - 6

    柯金龍星劉羽等。缺氧狀態下人顳下頜關節滑膜成纖維細胞環氧合酶2的表達及意義。中華醫學雜志， 2005 ， 85 （ 25 ） : 1747 - 1751
  15. They demonstrate that under hypoxic conditions mif activates the cellular stress response enzyme amp - activated protein kinase ( ampk ), which in turn is a key regulator of metabolism protecting against ischemic damage

    他們證實，在缺氧環境下mif激活細胞內應激反應酶腺苷酸活化蛋白激酶（ ampk ） ，它是隨后抗缺血損傷代謝調控的重要元素。
  16. Influence of polygonatum polysaccharide on apoptosis of primary cultured neonate rat cerebral cortical neurons caused by hypoxia

    黃精多糖對新生大鼠大腦皮層神經細胞缺氧性凋亡的影響
  17. Mild hypothermia attenuates neuronal apoptosis after cerebral hypoxiaischemia in neonatal rats

    亞低溫減輕新生大鼠缺氧缺血腦細胞凋亡的作用及機制研究
  18. To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

    為了驗證上述假設，本文分別在整體和離體水平，採用大腦中動脈缺血（ middlecerebralarteryocclusion ， mcao ）和體外氧?糖剝奪（ hypoxiaandglucose - deprivation ， hgd ）缺血模型，首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況，觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化，然後通過外源性給予vegf蛋白，在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響，最後利用vegf反義脫氧寡核苷酸（ oligodeoxynucleotide ， odn ）特異阻斷內源性vegf蛋白的表達，觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用，以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。
  19. Channel gating, ion selectivity, or ionic conductance and regulatory mechanisms of the ion channels on cell membranes. besides, we pay attention to the effect of intermittent and sustained hypoxia to the movement of ions. we also try to simulate an inflammatory environment to survey the changes in infection and trauma

    其中生理部分，在於研究細胞膜表面各種離子孔道的特性如離子孔道啟閉離子選擇性或離子通透度等和調控機制，同時並著重於間歇性與持續性缺氧的影響至於病理部分，則模擬感染或外傷的環境來觀察對離子運輸的改變。
  20. He hopes xtreme everest will confirm dr montgomery ' s work by showing that how well a person deals with hypoxia is related to the efficiency with which his cells utilise oxygen ? and that this, in turn, is genetically determined

    他希望嚴酷的珠穆朗瑪峰可以通過展示「一個人抵抗組織缺氧的能力和他細胞利用氧氣的效率有關系，而這往往是先天決定的」 ，來印證蒙哥馬利教授的理論。
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