肝細胞毒性 的英文怎麼說

中文拼音 [gānxìbāodúxìng]
肝細胞毒性 英文
hepatotoxicity
  • 肝 : 名詞（人和高等動物的消化器官之一） liver
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 毒 : Ⅰ名詞1 （對生物體有害的性質或物質; 毒物） poison; toxin 2 （毒品） drug; narcotics 3 （姓氏） a s...
  • 性 : Ⅰ名詞1 （性格） nature; character; disposition 2 （性能; 性質） property; quality 3 （性別） sex ...
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  • 毒性 : [藥理學] toxicity; virulence; poisonousness毒性測定 toxicity test
  1. Human augmenter of liver regeneration ( halr ) is a novel cytokine which stimulates specifically hepatic cell proliferation and is able to rescue acute liver failure caused by hepatotoxin for example carbon tetrachloride and galactosan amine et al

    人肝再生增強因子（ humanaugmenterofliverregeneration ， halr ）是一種新的細胞因子，能特異性地刺激肝源細胞的增殖，並對四氯化碳、半乳糖胺等肝毒劑引起的肝損傷有治療作用。
  2. Using cd, cu, zn and cr as experimental toxicants and crucian as experimental creature, we employed the environmental biotechniques ( flame atomic absorption spectrophotometry, the method of improved pyrogallol autoxidation and electron microscopy ) to study the acute toxicity and secure concentrations of heavy metals to crucian ; the accumulation and distribution of mixed heavy metals to fingling crucian ; the activities of superoxide dismutase ( sod ) of gill and liver tissues and the hispathological and ultrastructural change of superfine structure of liver and kidney of crucian after cadmium exposure. through which, we intended to fully and systemically study the toxic influence of heavy metals to fish, and preliminarily discuss the influence of heavy metals pollution to the diversified level of fish such as individual, organ, tissue, cell and molecule. thus we tried to provide scientific evidence to establish preventative management measures, avoid and relieve the harm of heavy metals pollution to aquicolous ecosystem in time

    隨后以這幾種重金屬作為實驗毒物，以鯽魚為實驗動物，應用環境生物技術、火焰原子吸收分光光度法、鄰苯三酚自氧化法、透射電鏡等技術研究了重金屬cu 、 zn 、 cd和cr對鯽魚的急性毒性及其安全濃度評價、混合重金屬在鯽魚幼體組織內的積累和分佈、 cd對鯽魚鰓和肝臟中sod活性的影響、 cd對鯽魚肝細胞和腎細胞超微結構的影響等，全面和系統地研究了水環境中重金屬暴露對魚類的毒性影響，初步探討了重金屬污染對魚類的個體? ?器官? ?組織? ?細胞? ?分子等各水平層次的影響，為制定漁業生產上預防性的管理措施提供科學依據，及時避免或減輕重金屬污染對水生生態系統造成的損害。
  3. Even so, by truncating hbv pres gene, we finally obtained some useful " " bailors ", either nontoxic or self - activating, and used them to fish dna fragments of hbv pres interacting protein ( s ) from an ad vector constructed human embryonic cdna library

    我們通過第回軍巨大學碩士學位論文對pres基因分段截短的方法，獲得了對酵母細胞即無毒性作用，又沒有自激活作用的「誘餌」 ，通過它在酵母雙雜交系統中篩選構建於ad載體的人胎肝。
  4. We found nuclear deformation lymphocytes with increased heterochromatin and impaired thymus epithelium cells with increased lysosomes and deformation of mitochondrias. ( 4 ) effects on mouse liver of so2 challenge : so2 can cause significant liver injury. he staining showed several kinds of necrosis of liver including spot necrosis, focal necrosis and submassive necrosis infiltrated with lymphocytes, monocytes, few neutrophils and eosinophils ; tem observation showed fatty degeneration with dispersion of fatty droplets and dilation of rough endoplasmic reticulums, acid degeneration with significant hyperplasia of mitochondrias, necrosis of hepatocytes with karyorrhexis and other organelles losing their normal structure

    （ 4 ）二氧化硫染毒對小鼠肝臟的組織學結構有明顯影響，可引起肝臟點狀壞死、灶狀壞死甚至片狀壞死，伴隨不同程度的炎性細胞浸潤；透射電鏡觀察發現二氧化硫可引起肝細胞脂肪變性、嗜酸性顆粒變和壞死，脂肪變肝細胞中可見大小不等的脂滴存在，嗜酸性顆粒變肝細胞中可見線粒體明顯增生，壞死肝細胞可見細胞核結構破壞，細胞器減少，細胞膜不完整。
  5. From different points of view of the epidemiology, cytogenetics, molecular biology, experimental pathology and preventive medicine, this study revealed and proved that the genotoxicity and carcinogenesis existed in the drinking pond water in the high incidence area of live cancer in guangxi, indicated the feasible preventive and interruptive methods, too

    從分析流行病學，細胞遺傳毒理學、分子生物學、實驗病理學和預防醫學的不同角度，揭示和論證了廣西某肝癌高發區居民飲用塘水的基因毒性和致癌性，提出了切實可行的預防和阻斷措施。
  6. Results in the 260 slices of liver biopsy, there were 20 ones of acute viral hepatitis, 78 ones of chronic hepatitis, 79 ones of hepatocirrhosis, 28 ones of primary liver cell cancer, 27 ones of liver karyon heterogeneity, 20 ones of liver metastatic adenocarcinoma, 12 ones of fatty liver, five ones of alcohol liver, three ones of liver cyst, two ones of congenital bile duct atresia, six ones of illegible structure, 20 ones without liver cell or with few scattered liver cell

    結果260例肝穿活檢組織病例中，急性（病毒性）肝炎20例；慢性肝炎78例；肝硬化39例；原發性肝細胞癌28例；肝細胞核異質27例；肝轉移性腺癌20例；脂肪肝12例；乙醇肝5例；肝囊腫3例；先天性膽管閉鎖2例；穿刺組織結構模糊6例；未穿到或僅穿到少量肝細胞20例。
  7. Phenotypes and functions of dendritic cells derived from peripheral blood monocytes of chronic hepatitis b patients with different hbv dna loads

    不同病毒載量的慢性乙型肝炎患者外周血樹突狀細胞的表型和功能
  8. Containing many t cell epi - positions and neutralizing antibody epi - positions, the core protein region and envelope protein 2 of hepatitis c virus ( hcv ) structural protein may induce protective immunity responsion in body

    丙型肝炎病毒（ hepatitiscvirus ， hcv ）結構蛋白的核心蛋白（ c ）和包膜蛋白2 （ e2 ）含有多個t細胞表位和中和抗體表位，可誘導機體產生保護性免疫應答。
  9. To modify lysine with 2, 4, 6 - trinitrobenzene sulphonic acid accompanied with the decrease of haemagglutination and raitogenic activity both

    進一步用hepg2 . 2 . 1 . 5細胞作為乙型肝炎病毒模型，研究了黃精凝集素h的抗hbv活性。
  10. Chronic hepatitis b tcm syndrome type peripheral blood mononuclear cells

    慢性乙型肝炎中醫證型外周血單個核細胞乙肝病毒脫氧核糖核酸
  11. Individual hepatocytes are affected by viral hepatitis

    病毒性肝炎單個肝細胞受累。
  12. Viral hepatitis leads to liver cell destruction

    病毒性肝炎導致肝細胞破壞。
  13. Conclusion : for hcc patients with hb chronic infection / exposure, a high iral load prior to treatment is an aderse factor for surial and may be associated with a higher incidence of seere hepatitis during chemotherapy

    結論:有慢性乙肝感染或接觸史的肝細胞性肝癌患者，化療之前有高病毒負荷量是其生存的負面因素，並與化療期間急性肝炎的高發生率相關
  14. Future strategies to improe the prognosis of hcc patients undergoing chemotherapy should consider supportie therapy that incorporates antiiral therapies to reduce hb iral load

    將來要改善肝細胞性肝癌化療患者預后的措施應考慮同時給予減少病毒負荷量的抗病毒輔助治療
  15. Chronic hepatitis - prolonged inflammation and death of liver cells persisting for six months or more caused by hepatitis viruses, autoimmune factors, and on occasion, unknown conditions

    慢性肝炎? ?長期發炎和肝細胞壞死，持續6個月以上，由肝炎病毒引起，或由自體免疫引起，有時原因不明。
  16. Release of infectious irus particles was confirmed, as media from these cells were able to infect naie hepatocytes

    大量感染性病毒顆粒釋放，然後又感染新生的肝細胞。
  17. After yellow aspergillus toxin enters the person ' s liver, be released to issue the part that has active by splitting decomposition, action at liver cell, have the effect that cause cancer

    黃麴黴毒素進入人的肝臟后被裂解釋放出具有活性的成分，作用於肝細胞，具有致癌作用。
  18. It has been showed that hepatitis b virus ( hbv ) surface antigen pres, an important protective antigen, possesses many biological functions, and thus is considered to be closely associated with establishment of hbv infection, hbv replication and assembly in hepatocytes

    Hbv表面抗原前s區具有豐富的生物學功能和較強的免疫原性，前s區與hbv對肝細胞的感染、病毒復制及病毒顆粒的裝配密切相關，是hbv重要的保護性抗原。
  19. The recombinant virus adhuctla4 - ig prepared in this study efliciently inltcted l - o2 " cells, and the infected cells expressed a - nd excreted soluble rccolllbina11t protein huctla4 - ig

    該重組病毒在體外能有效感染正常肝細胞株l - o2 ，受感染細胞能表達、分泌可溶性的重組融合蛋白huctla4 - ig ； 2
  20. Hepatitis b is caused by a highly contagious virus that infects the liver

    乙肝是由一種高度傳染性的嗜肝細胞病毒感染所致。
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