脈動活動核 的英文怎麼說

中文拼音 [màidònghuódònghé]
脈動活動核 英文
pulsating active nucleus
  • 脈 : 脈名詞1. （動脈和靜脈的統稱） arteries and veins2. （脈搏的簡稱） pulse 3. （像血管的組織; 連貫成系統的東西） vein
  • 活 : Ⅰ動詞1 （生存; 有生命） live 2 [書面語]（救活） save （the life of a person）:活人無算 （of a goo...
  • 核 : 核構詞成分。
  • 脈動 : [物理學] [天文學] pulsation; pulsing; pulse; ripple; impulsive motion; pulsating; panting; loping...
  • 活動 : 1 （運動） move about; exercise 2 （動搖） shaky; unsteady 3 （不固定） movable; mobile; flexible...
  1. Based on the analysis of thermal contact metamorphic aureoles, granitoid interior structure, quartz axis fabric and strain characters in the different rock unites of yuanshishan granitoid and its wall rocks, the emplacement mechanism of yanshishan granitoid was studied. the magmatic explictite dynamic was advanced in light of regional stress and magma dynamic research. those conclusion was synthesized to ascertain the emplacement mechanism - granitic magma ascended in dikes with the minimum critical width of the magma ascending channels about 2. 213. 88m and located in the core of yindianshan dome with the passive style of dyke explictite ; the later unite is a kind of multiple emplacement mechanism with the character of active emplacement of ballooning

    同時運用巖漿動力學原理從另一個角度探討其侵位驅動力。綜上指出：經分凝后的活動性巖漿在深部側向擠壓和浮力的聯合驅動下，沿深大斷裂以最小臨界寬度為2 . 213 . 88m的裂隙脈動上侵，整體以巖墻擴展的方式被動定位於銀殿山穹窿的核部，但後期屋面前單元具有主動侵位的特點應為一種復合定位機制。
  2. Tumor necrosis factor ( tnf ), a cytkine that exerts many pro - atherosclerotic effects in vivo, causes up - regulation of acat - 1 gene expression in human blood monocytes. by luciferase activity assay, tnf - a enhanced acat - 1 p7 promoter activity in thp - 1 monocytic cell line

    細胞因子tnf -即腫瘤壞死因子大量存在於人動脈粥樣硬化斑塊中，本實驗通過熒光素酶活性和rt - pcr測定結果表明， tnf -處理人血單核細胞和單核細胞株thp - 1均增強acat - 1基因p7啟動子活性。
  3. A mononuclear inflammatory cell infiltrate extends from portal areas and disrupts the limiting plate of hepatocytes which are undergoing necrosis, the so - called " piecemeal " necrosis of chronic active hepatitis

    單核細胞浸潤從門脈區延伸，破壞正在壞死（也稱為慢性活動性肝炎的「碎片狀」壞死）的肝細胞界板。
  4. The satisfying demand of customers " needs is responded through the aspects in quality, cost, delivery time limit etc. a prosperous development structure is formed. that is product with no adventure, development with no wasting, and production with no wasting. thus it is very helpful to improve management of enterprise activity and increase enterprise vigor, but the more important, consciousness of staff is basically altered to use effective way in enterprise, regard manpower as core, increase consciousness of the whole participation, construct a system to prevent disaster harmful trends and wasting from happening and facilitate the change of management conception at the same time

    本文介紹了建設集團在2000年度下半年導入tpm活動（全員參加的生產經營改善活動） ，確定了質量、設備、生產、技術、成本、安全等六個方面的推進方向，在銷售、開發、購買、製造各個環節進行了實施，整個改善活動實施中以組織為脈絡，以現場為背景，以人力為核心，以管理為手段，以產品為結果作為切入點，把開發、生產及銷售過程作為企業生產經營改善的突破口，從質量、成本、交貨期等方面及時響應客戶所需產品的滿意要求，使公司形成了市場需要風險為「零」的產品，開發浪費為「零」 ，生產浪費為「零」的格局。
  5. To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

    為了驗證上述假設，本文分別在整體和離體水平，採用大腦中動脈缺血（ middlecerebralarteryocclusion ， mcao ）和體外氧?糖剝奪（ hypoxiaandglucose - deprivation ， hgd ）缺血模型，首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況，觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化，然後通過外源性給予vegf蛋白，在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響，最後利用vegf反義脫氧寡核苷酸（ oligodeoxynucleotide ， odn ）特異阻斷內源性vegf蛋白的表達，觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用，以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。
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