Objective to report 16 cases of intracranial aneurysms with calcified vascular wall and study the relationship between vascular calcification and pathogenic mechanism of intracranial aneurysms. methods spiral ct scan, clinical data and serum calcium, phosphate, glucose, lipid, renal function of the 16 patients were studied in retrospect. all aneurysms were clipped, in which 8 were resected after clipping. results the location of aneurysms with aneurismal wall calcification was, on posterior communicating artery in 3, on middle cerebral artery in 2, on anterior communicating artery in 2, on basilar artery in 5 and on basilar artery in 4. all patients showed normal serum calcium, phosphate, glucose and normal renal function. serum lipid of patients had been elevated. all patients recovered with no significant neurological deficits. conclusion intracranial vascular calcification is an active and regulated process in close relation to atherosclerosis, serum calcium, phosphate, lipid, thyroid, parathyroid function and other factors. intracranial vascular calcification has close relationship with the pathogenesis and management of intracranial aneurysms
目的報告16例瘤壁有鈣化的顱內動脈瘤病例的治療，分析顱內血管鈣化與顱內動脈瘤病理發生機制的關系.方法回顧性分析16例瘤壁有鈣化的顱內動脈瘤病例的影像學、臨床資料及手術治療.結果瘤壁有鈣化的顱內動脈瘤的位置是: 3例位於后交通動脈， 2例位於大腦中動脈， 2例位於前交通動脈， 5例位於椎動脈， 4例位於基底動脈.所有患者血鈣、磷酸鹽、血糖、
腎功能正常. 6例患者血脂增高. 16例患者均行動脈瘤
夾閉術，其中8例術中切除動脈瘤，治療效果良好.結論動脈瘤瘤壁鈣化與其病理發生機制有關，常見于巨大或大動脈瘤，瘤壁血管鈣化是與動脈粥樣硬化、血鈣、磷酸鹽、血脂、甲狀腺、甲狀旁腺等因素有關的主動耗能，多因素調控的病理過程
A experimental study on effect and mechanism of reducing the blood pressure with two kinds of moxibustion therapies
兩種艾灸法對二
腎一
夾型高血壓大鼠血壓和血管內皮細胞內分泌功能的影響
It is argues that aiha roots from spleen and kidney deficiency, and exhibits the internal accumulation of dampness, heat, blood stasis and toxin ; and that its treatment focuses on both the root and branch aspects, supporting healthy qi and removing pathogenic factors
認為該病的中醫病機特點是脾腎虧虛，濕、熱、瘀、毒內伏，本虛標實，虛實夾雜；臨證治療宜扶正祛邪、標本兼顧。
