血細胞管型 的英文怎麼說

中文拼音 [xiěxìbāoguǎnxíng]
血細胞管型 英文
blood cast
  • 血 : 血名詞（血液 多用於口語） blood:吐血 spit （up） blood; 血的教訓 a lesson paid for [written] in b...
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 管 : Ⅰ名詞1 （管子） pipe; tube 2 （吹奏的樂器） wind musical instrument 3 （形狀似管的電器件） valve;...
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  • 管型 : cast
  1. Section two the evaluation of biocompatibility of the acellular dermal matrix by the method of cell culture. the new born rat ' s epdermic cells were cultured with the acellular dermal matrix together as experiment group, while the epdermic cell were cultured simply as control. 24 hours later, under the invert microscope, the epidermic cells anchored well and transparent flat cells were observed in both groups. 7 days later, both cultured cells were taked out and fixed in 95 % ethanol, stained with hematoxylin and were observed under light microscope. many cleaved cells were observed in both groups. during cell culture, no pathogenic microganism was observed. so we considered the acellular dermal matrix was aseptic and had good biocompatibility. section three subdermal implantation of the acellular dermal matrix. 24 rats were used in the experiments. a piece of acellular dermal matrix ( 1. 5 x 1. 5cm2 ) was implanted beneath the dorsum skin flaps of each rat, 1 week, 2 weeks, 3 weeks and 4 weeks after implantation, 6 pieces of acellular dermal matrix were harvested and the size of implanted acellular dermal matrix were measured, the sections were used for he staining and observed under light microscope. the result were as folio wing : 1 - 2 weeks after implantation, the acellular dermal matrix began to adhere to the tissue around and turned red gradually ; 3 - 4weeks after implantation, the acellular dermal matrix adhered closely to the tissue around and could be recognized easily, 1 - 3 weeks after implantation, the size of implanted acellular dermal matrix had no statistical difference ( p > 0. 05 ). 4 weeks after implantation implanted acellular dermal matrix contracted ( p < 0. 05 ). under light microscope, l - 2weeks after implantation, the fibroblast cells infiltrated the acellular dermal matrix and a small amount endothelial cells of vessel and lympho - histiocytic cells infiltrated the acellular dermal matrix. 3 - 4 weeks after implantation, infiltrating blood vessels were evident. so we think that the acellular dermal matrix had low immunological reactions and could induce the infiltration of fibroblast macrophage cell and the endothelial cells of vessel

    結果如下：皮下包埋卜周者，無細胞真皮基質漸與周圍組織粘附，顏色由蒼白轉紅；皮下包埋3周者，無細胞真皮基質與周圍組織緊密枯附，盾晰葉辯；術后卜周，包埋的基質面積變化較包埋前無統計學差異o川0引，術后4周包埋的無細胞真皮基質面積較包埋前縮小j刃刀5 ） 。光鏡下術后卜周，宿主的淋巳組織細胞、成纖維細胞浸入生長，釉附在膠原纖維上，少量血管內皮細胞浸入基質；術后34周，無細胞真皮基質內較多的血管形成，故可認為無細胞真皮基質免疫原性低，能誘導宿主的成纖維細胞、巨噬細胞浸入生長，為一種新型的真皮替代物。第四部分無細胞真皮基質與自體斷層皮片復合移棺的研究， sd大鼠10隻，在其背部卜方造成全厚皮膚缺損的創面
  2. However, they are anastomosing and not collapsed, and are accompanied by atypical endothelial cells, eosinophilic hyaline globules, plasma cells and fascicles of spindle cells

    然而，它們的血管網是吻合的，不塌陷，伴不典型內皮細胞、嗜堿性玻璃樣小球、漿細胞和梭形細胞束。
  3. This article reviews the clinical manifestations, mutation feature, gene location and phenotype of different ischemic cerebrovascular disease caused by monogenic disorders, including coagulation disorders, erythrocytic disorders, inherited small vessel disease, metabolic disorders, connective tissue diseases, vasculopathies and disorders of unknown etiology

    本文主要闡述了單基因遺傳障礙引起的缺血性腦血管病，包括凝血障礙、血細胞病、遺傳性小血管病、代謝障礙、結締組織病、大動脈病及不明原因引起缺血性腦血管病的臨床特徵、突變特點、基因定位及表型等遺傳學研究進展。
  4. The steady dead generation and time that was caused by the isolated virus was certain by chicken embryo which was inoculated on seven or nine days. the histopathological changs of the infectious stunting syndrom were studied by the way of ordinary paraffin section and he dying. the experimental result were as follows : the test proved that the changes of the chicken embryo were different in different stage. the chicken embryo dead in a week after it inoculated. the body was dropsy and hemorrhage. dead before it hatched out, the embyo body were dropsy, pale and slime. the liver was yellow and swolled, gallbladder ( vesica fellea ) was filled with bile. bursa and glandula thymus analosis. the kindey dropsy. bowel lamina were humble, dilatation. gas and yellow foam were filled the bowel. histopathological changes were that, in early stage, obvious changes of liver and kindey were dropsy, hemorrhage and necrosis. two types eosinophilic intranuclear inclusion bodies including large round and little granular were present in cells of the above organs. the obvious changes of bursa were dropsy, adverse folliiculated growth and little lymphocytes proliferating, 19 - 21 days chicken embryo, one or two big empty vacuoles were prensent in cells of liver and kindey. the number of the folliculi was growing, the vacuoles between cells were larger

    膽囊充盈、其內充滿稀薄的膽汁；法氏囊、胸腺萎縮，腸道擴張、腸壁菲薄、內充滿氣體及黃色泡沫狀物；腎臟腫大。病理組織學變化方面，早期肝臟、腎臟、腸主要以出血、水腫和壞死為主，且肝細胞核及腎小管的上皮細胞核內均發現有核內包涵體，包涵體呈嗜酸性，為大型圓形包涵體或不規則的顆粒狀；法氏囊則以水腫、濾泡發育不良、小型淋巴細胞數量增多為主。 19 21日齡雞胚肝細胞、腎小管上皮細胞的胞漿內出現1 2各大的空泡，法氏囊濾泡數目增多細胞間有較大空隙。
  5. The x - rays allowed the researchers to look at the distribution of copper in both a cell model of angiogenesis and sections of breast tumor tissue rich in blood vessels

    線可以讓研究人員在血管生發的一個細胞模型和有豐富血管的乳腺癌組織中都可以看到銅的分佈。
  6. The purposes of the present study were to investigate ( 1 ) the hemodynamic effects of agmatine in anaesthetized dahl salt - sensitive ( ds ) hypertensive and dahl salt - resistant ( dr ) rats ; ( 2 ) the effect of agmatine on vascular tension in the isolated aortic artery of rats and the underlying receptor mechanism ; ( 3 ) the effects of local injection of agmatine on femoral, renal, and mesenteric vascular beds by constant flow perfusion method ; ( 4 ) the effect of agmatine on l - type calcium current ( / ca - t ) in rat ventricular myocytes with whole - cell configuration of the patch - clamp technique ; ( 5 ) the effects of agmatine on free intracellular calcium concentration ( ca2 + d of isolated rat ventricular myocytes

    （ 3 ）採用後肢、腎臟和腸系膜動脈在體恆流灌注法，觀察向灌流環路中直接注射胍丁胺的血管效應。 （ 4 ）應用全細胞膜片箝技術，觀察胍丁胺對大鼠心室肌細胞l -型鈣通道電流（ i _ （ ca - l ） ）的影響。 （ 5 ）用fluo3 - am負載分離的大鼠心室肌細胞后，由激光共聚焦法測定單個心室肌細胞[ ca ~ （ 2 + ） ] _ i的熒光強度，觀察胍丁胺對分離大鼠心室肌細胞內游離鈣濃度（ [ ca ~ （ 2 + ） ] _ i ）的影響。
  7. Active programs include tissue engineering models for cardiac muscle, tri - leaflet valves, cell - based cardiac pumps and vascular grafts

    熱點項目包括心肌組織的組織工程模型、三葉瓣、細胞型的心臟泵及血管移植物。
  8. Ganoderma lucidum polysaccharides inhibit angiogenesis and cell adhesion in chicken chorioallantoic membrane model

    靈芝多糖抑制雞胚尿囊膜模型中的血管生成及細胞黏附
  9. No, a first gas information molecule discovered in human being, is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels. in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated. in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no, the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation, both of which are demonstrated to induce the

    在內毒素休克過程中vec是lps及其誘導機體產生的多種促炎細胞因子如tnf 、 il - 1作用的主要靶細胞， vec誘導型一氧化氮合酶（ induciblenitricoxidesynthase ， inos ）激活、 no大量誘生而內皮型一氧化氮合酶（ endothelialnitricoxidesynthase ， enos ）活性被抑制、 no生成障礙，是血管反應性異常變化、血管調節機制紊亂的重要發病環節。
  10. This paper expounds the progresses of the researches on the life science and relevant subjects, which include the research of cultivating the arterial vessel with fine cell tissue, the research on the genetically modified animal, the research on the pest control with bio - virus pesticide, the research on the death mechanism of protein and the diseases of immune system ( including cancer ), the research on anti - senile protein and the research on compound - type aids vaccine

    闡述了生命科學及其相關學科的研究進展，包括用細小細胞組織培製成動脈血管的研究、轉基因動物的研究、生物病毒農藥防治蟲害的研究、蛋白質死亡機理及免疫系統疾病（包括癌癥）的研究、抗衰老蛋白的研究以及復合型艾滋病疫苗的研究。
  11. Study on the secretion of endothelial cells in rabbits with blood hyperviscosity and intervention effect of recipe of promoting the blood circulation and removing blood stasis

    高黏滯血癥模型兔血管內皮細胞分泌功能變化及活血化瘀中藥干預作用研究
  12. We also investigated the pathological changes of mouse liver, thymus and cerebrum cortex challenged by so2 inhalation by in vivo tests. we studied the apoptotic induction on mouse spleen cells and cytotoxicity of human embryo lung fibroblasts of so2 derivatives by in vitro tests. in vivo tests of sulfur dioxide inhalation showed : ( 1 ) effects on mouse lung of so2 challenge : we found no significant apoptotic changes induced by so2 inhalation but obvious pathological changes of lung with vacuolating of osmiophilic multilamellar bodies which maybe related with the decrease of surfacant and decrease of microvillus of type ii alveolar cells ; we also found thickening of part of basement lamina between type i alveolar cells and capillary endothelium cells which may inhibit the dispersion of oxygen and contribute to lung dysfunction

    二氧化硫熏氣染毒的體內實驗結果表明，在本次實驗的濃度范圍內（ 56mg m ~ 3 、 112mg m ~ 3 、 168mg m ~ 3低、中、高三個濃度） ： （ 1 ）通過透射電鏡、 dna凝膠電泳分析和流式細胞分析發現二氧化硫吸入染毒一周對小鼠肺臟沒有明顯的凋亡誘導作用，但通過透射電鏡觀察發現二氧化硫可引起肺臟明顯的超微結構改變，引起型肺泡上皮細胞板層體空泡化，微絨毛減少，線粒體緻密化或腫脹變性；肺泡血管內皮細胞和型肺泡上皮細胞之間基膜增厚，使氧氣彌散功能出現障礙，從而降低肺功能。
  13. A experimental study on effect and mechanism of reducing the blood pressure with two kinds of moxibustion therapies

    兩種艾灸法對二腎一夾型高血壓大鼠血壓和血管內皮細胞內分泌功能的影響
  14. Effect of electroacupuncture on ultrastructure of hippocampal neuroglial cells and capillaries in vascular dementia rats

    電針對血管性癡呆模型大鼠海馬神經膠質細胞及微血管的影響
  15. Methods to extend the migration of endothelial cells on sprouts from four directions - moving up, down, left or right - in previous models to random direction in the present model, and develop a relevant 2d discrete model to simulate the process of tumor angiogenesis and compare the simulation results with some corresponding experimental data

    方法將新生血管芽尖的內皮細胞遷移方向由先前模型的上、下、左、右四個方向擴展為任意方向，建立相應的二維離散模型，數值模擬實體腫瘤微脈管系統的生成過程，比較模擬結果與相關實驗數據的符合程度。
  16. This illustration ( same case as preious image ) shows endothelial cells arranged in more compact sheets with abortie ascular spaces. moderate cytologic atypia is seen

    圖中可見內皮細胞呈顯緊密的片狀排列，頓挫性血管間隙，中度細胞非典型性。
  17. Course contents ( containing practice period ) : introduction to the course ; basic function of cells ; breath ; blood ; blood circulation ; digesting and absorption ; energy metabolism and body temperature ; the excretion of kidney ; sensory organs ; nervous system ; endocrine system ; urogenital system ; physical constitution ; change of physiological function of human body in each sports stage ; lung ' s function measurement ; blood type test ; hemoglobin measurement ; auscultation of heart sounds ; measurement of blood pressure ; electrocardiogram measurement ; measurement of visual field ; measurement of body temperature ; cardiovascular function test before and after sport

    課程內容（含實踐活動） ：緒論、細胞的基本功能、呼吸、血液、血液循環、消化與吸收、能量代謝和體溫、腎臟的排泄、感覺器官、神經系統、內分泌、生殖、身體素質、運動各階段人體生理功能的變化、肺功能測定、血型鑒定、血紅蛋白測定、心音聽診、血壓測定、心電圖測定、視野測定、體溫測定、運動前後心血管功能變化。
  18. Result : the layers of tissue engineeringed vascular model were combined tightly. hvec, vsmc and fibroblast can secrete extracelluar matrix ( ecm ) and interchange substance, resemblin physiological functions

    構建后的血管模型，層與層結合緊密，內皮細胞、平滑肌細胞及成纖維細胞分泌細胞外基質，相互進行物質交換，類似於生理條3第四軍醫大學碩士學位論文件。
  19. The following research areas will be involved in the project : ( 1 ) mechanism of hereditic factors and its function in chinese type 2 diabetes ; ( 2 ) metabolomics on the cell stress and its nutrition intervention in the type 2 diabetes ; ( 3 ) mechanisms of metabolic nuclear receptors in the process of insulin resistance and type 2 diabetes onset ; ( 4 ) roles of signal transduction molecules, lipid metabolic disorder to insulin resistance ; ( 5 ) type 2 diabetes development and role of pathological changes of blood vessels ; ( 6 ) abnormal insulin secretion, apoptosis and type 2 diabetes ; ( 7 ) function of molecular network of type 2 diabetes progression

    本項目擬開展以下研究工作： （ 1 ）中國人2型糖尿病發生過程中遺傳因素的發現及其作用機制； （ 2 ）細胞應激在中國人2型糖尿病發生過程中的作用機制及其營養干預的代謝組學； （ 3 ）代謝性核受體在胰島素抵抗和2型糖尿病發病中的作用機制； （ 4 ）細胞信號轉導分子、脂代謝紊亂與胰島素抵抗的關系； （ 5 ） 2型糖尿病發展過程與血管病變； （ 6 ）胰島素分泌異常、細胞凋亡與2型糖尿病； （ 7 ） 2型糖尿病發生過程中的分子網路與作用機制。
  20. Based on poor glandular formation, necrosis, cellular atypia, nuclear pleomorphism, invasion of soft tissue and bone, and vascular permeation

    主要基於腺管的結構分化不良，壞死，細胞異型性，核多型性，軟組織和骨浸潤以及血管浸潤。
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