調制抑制 的英文怎麼說

中文拼音 [diàozhìyìzhì]
調制抑制 英文
modulation suppression
  • 調 : Ⅰ動詞1 （配合得均勻合適） harmonize; suit well; fit in perfectly 2 （使配合得均勻合適） mix; adju...
  • 制 : Ⅰ動詞1 （製造） make; manufacture 2 （擬訂; 規定） draw up; establish 3 （用強力約束; 限定; 管束...
  • 抑 : Ⅰ動詞（向下按; 壓制） restrain; repress; curb Ⅱ連詞[書面語]1 （表示抉擇） or 2 （表示轉折） but3 ...
  • 抑制 : 1 （控制） restrain; control; check; hold up; curb; stop; repress; bridle; choke; prehension; sup...
  1. Not only the neurons but also the as in the rat brain play a very important role in the process of the peripheral nociceptive stimulation ; ? he newly - found eda may be the functional intercommunication ultrastructure between neurons and as, through which the as could modulate the neurons in the pain signal processing ; @ as might play an active and important role for modulation of pain through a new pathway from the peripheral afferent nerve to the as, then to neurons via electron - dense areas forming the glia - neuron s ignaling network

    光、電鏡水平發現as與神經元之間有多種途徑進行信息交流，新發現的eda有可能是神經元與as之間信息交流的結構之一。阻斷as與神經元間信息交流連接后，前者對外周組織4第四軍醫大學博士學位論文損傷所致疼痛的反應不受影響，而後者的反應明顯受到抑制，提示在非病理性疼痛反應中，膠質細胞對神經元具有主動調控作用。
  2. Time grew, the adjustment that can bring about pallium again the function is disorder, parasympathetic nerve centre is met " seize the opportunity " come out from the disengagement below the control of pallium, stimulant increase, the alvine path exercise that gets it to control is accelerated, patient occurrence diarrhoea ; this nerve excitement is too long, but because of " fatigue " and ebb gradually and turn to restrain, alvine path muscle also comes down flabbily slowly, peristalsis is abate, the result causes constipation again

    時間長了，又可導致大腦皮層的調節功能紊亂，副交感神經中樞便會「乘機」從大腦皮層的控制下解脫出來，興奮性增強，受它支配的腸道運動加快，病人出現腹瀉；該神經興奮過久，可因「疲憊」而逐漸衰退並轉為抑制，腸道肌肉也就慢慢地鬆弛下來，蠕動減弱，結果又引起便秘。
  3. Under mixed culturing conditions, it was observed that bacterial number rapidly incre ; ised soon after the lysing of host cells. on the contrary, while the non - host cyanobacterium ( i. e. anabaena flos - aquae ) was incubated in the mix culturing system, the breeding of the bacteria could be inhibited. it may be suggested from the result above that cyanophage could bring about the changes in microorganism populations

    調查結果顯示， 19個採集的水樣中有6個含有裂解織線藻的噬藻體，而且水樣未經濃縮即能裂解宿主，說明噬藻體在淡水中分佈較廣泛，裂解性也較強；混合培養條件下的研究結果表明，噬藻體裂解宿主后，細菌數量快速增加，而當培養系統中有非宿主藻類存在時，細菌的增殖則受到非宿主藻的抑制，說明噬藻體可以顯著改變系統中微生物的種群結構。
  4. T cell will be apopotosis, clone depletion and / or anergy, in turn inducing an antigen - specific tolerance. ctla4 ( cytotoxic t lymphocyte associated antigen 4 ), a prominent negative costimulator, can competitively bind with b7, and block b7 - cd28 pathway and t cell activation by producing inhibitory singals

    Ctla4 （ cytotoxictlymphocyteassociatedantigen4 ）作為一種主要的共刺激負調節劑，可競爭性結合b7分子，阻斷b7 - cd28途徑、並通過產生抑制性信號使t細胞活化障礙。
  5. Transplantation has been developed to an effctive strategy for treatment of end stage diseases of multiple organs since 1950 ' s. despite success of current immunosuppressive drugs, transplantation immunologists and surgeons are still seeking to achieve tolerance to allograft. there are several different mechanisms such as clonal deletion, anergy, neglect, immuno regulation. which maintain tolerance to self - antigen. some strategies, derived from these mechanisms has been proved to be encouraged in suppression of allograft rejection

    二十世紀五十年代以來，器官移植的進展使同種異體器官移植成為治療多種器官終末期疾病的有效手段。盡管目前免疫抑制方案非常有效，但免疫耐受仍是移植免疫學家和外科醫生關注的課題。有多個機制使免疫系統對自身抗原保持耐受，如：克隆清除、忽略、失能、免疫調節。
  6. At rest, the heterodimeric rel / nf - k b complex is located in the cytoplasm bound to an inhibitory factor, i k b. upon stimulation, i k b is phosphorylated and degraded, free nf - k b then translocates into the nucleus where it binds to k b site to regulate transcription

    靜息時， re皿4bh聚體與抑制蛋白kbs結合，在細胞漿中保持無活性。受到刺激， ikb分子迅速磷酸化並降解，釋放nf兒b轉位入核，通過與kb位點結合調節轉錄。
  7. The degradation of i k b and dissociation from nf - k b complex are essential process for activation of nf - k b. there is no report about whether cck - 8 inhibits nf - k b activity through inhibiting lps - induced degradation of i k b. the present study was designed to observe the regulatory effect of cck - 8 on changes of immune functions induced by lps, the receptor mechanism and signal transduction mechanism involving mapk, stat3 and i k b at different levels including in vivo and in vitro, in order to study its immunomodulatory effect on reversing es and its molecular mechanisms

    Ikb的降解、並從w兒b復合物上解離是w4b激活的必需過程。 ccks是否通過抑制lps誘導的ikb降解，從而抑制nf幾b激活尚未見報道。本研究在整體、細胞及分子水平，觀察了ccke對lps引起的免疫功能改變的調節作用，研究了其受體機制及mapk 、 stat3和ikb的信號轉導機制，以探討cck七抗es時的免疫調節作用及其分子機制。
  8. The insulin - like growth factor - 2 ( igf - 2 ) and igf conjugated protein - 6 ( igfbp - 6 ) mrna level in rat calvaria bone tissue and mc - 3t3 - el cells were detected by northern blotting analyses and reverse transcription polymerase chain reaction. the estrogen responsive element ( ere ) in igfbp - 6 gene promoter was identified and involved in tcdd - reduced regulation of the gene expression by electromobility shift assays ( emsa )

    正常胎鼠頭蓋骨組織igf一2mrna呈高水平表達狀態，而igfbp一6mrna的水平較低; ccf胎鼠頭蓋骨骨組織內igf一2mrna的表達較正常胎鼠降低， igfbp一6mrna的表達則明顯升高; atra和e :聯合應用時， atra可以抑制雌激素對細胞內igf一2和igfbp一6的這種調節作用。
  9. The protein nucleic acid complexes were identified by autoradiography for 48 h. the psg5heo and igfbp - 6 promoter pcat vectors were transfected into cos - 7 cells with iipofectamin2000 ; these cos - 7 cells where treated with 1 jxm e2, with lonm tcdd and with both lonm tcdd and 1 jxm e2, individually ; the cat protein was examined by cat - els a. results : the congenital skeletal defects in fetal rats were induced with 10 - 15ug / kg tcdd or with 100 - 140 mg / kg ra with dosage - dependence effect. the igfbp - 6 mrna in rat calvaria tissue and mc - 3t3 - el cells were increased with tcdd and ( or ) ra, whereas the cell proliferation and igf - 2 mrna in mc - 3t3 - el cells was decreased in the status of e2

    結果: tcdd在大鼠胚胎發育時期具有骨骼致畸作用，骨骼發育畸形包括:內翻足、脊柱裂、顱面骨發育缺陷、無尾畸形等;隨著tcdd濃度的升高（ 10一15林擴kg ） ，致畸率明顯上升（ 14 . 7 %一24 . 6 % ） ; tcdd和ra聯合用藥可以增強對大鼠骨骼發育的致畸效應; e :對成骨細胞內igf一2基因有正性調節作用，對igfbp一6基因有負性調節作用， tcdd可以抑制e :對細胞內xgf一2和igfbp一6的調節作用。
  10. This review and our recent studies suggest that : ( 1 ) baroreceptor reflex is blunted in heart failure ; ( 2 ) central angiotensin ii and reactive oxygen species play an important role in blunted baroreceptor reflex ; ( 3 ) cardiac sympathetic afferent stimulation and chemoreceptor reflex inhibit baroreceptor reflex ; and ( 4 ) exercise training normalizes abnormal reflexes in the heart failure state

    本綜述及我們近年的研究表明： （ 1 ）在心力衰狀態下壓力感受性反射功能明顯減弱（ 2 ）中樞血管緊張素和活性氧在壓力感受性反射功能失調中發揮關鍵作用； （ 3 ）心交感傳入刺激和化學感受性反射能抑制壓力感受性反射； （ 4 ）適當的運動可以部分糾正異常的心血管反射活動。
  11. Chemotaxis mediated by chemokine receptors, such as cxcr4, play a key role in lymphocyte homing and hematopoiesis as well as in breast cancer metastasis. we have previously demonstrated that ? - arrestin2 functions to attenuate cxcr4 - meidated g protein activation and to enhance cxcr4 internalization. here we further show that expression of ( - arrestin2 in both hela and hek293 cells significantly enhanced the chemotactic efficacy of stromal - cell derived factor 1 ( ( sdf - 1 ( ), the specific agonist of cxcr4

    - arrestin2是趨化因子受體的一種重要的調節蛋白，本研究工作發現在hek - 293或hela細胞中升高- arrestin2的表達水平會顯著增強cxcr4介導的趨化作用，反之當- arrestin2的表達被它的反義核苷酸或rnai所抑制， cxcr4介導的趨化作用則被明顯抑制。
  12. The effect of two types of emotion regulation, suppression and reappraisal, on dyadic interactions was investigated in the present study

    摘要本研究探討配對互動中情緒抑制和情緒重評兩類情緒調節的效應。
  13. The end product inhibitor has no structural similarity to the substrate of the enzyme it is regulating.

    最終產物抑制劑與它調節的酶的底物在結構上無相似之處。
  14. It has reported that the amygdaloid complex exerts ianuence on the medial geniculate body and the auditory cortex. not only the light evoked response of latera geniculate cells can be modulated by the amygdaloid complex, but also their receptive field characterstics. the pathway of this modulating innuence is amygdala isual cortexateral geniculate

    已有的工作表明，杏仁復合體可對內膝體和聽皮層聽覺信息產生影響作用，在視覺上，杏仁復合體不僅對兔外膝體神經元閃光刺激反應活動產生抑制和易化影響，而且對外膝體神經元的感受野特性具有調製作用，這種作用是通過杏仁-視皮層-外膝體途徑而實現。
  15. Protein kinase a is camp - dependent kinase, one of the most important signal transduction pathways, plays a pivotal role in growth, differentiation, tumor occur, cell cycle control, etc. pka activators such as camp, 8 - br - camp or phosphodiesterase ( pde ) inhibitor isomethyl butyl xanthine ( imbx ) or purified pka catalytic subunit all can inhibit germinal vesicle breakdown ( gvbd ) and meiotic maturation in mouse oocytes, also in xenopus oocytes

    蛋白激酶a （ proteinkinasea ， pka ）是依賴于camp的絲蘇氨酸蛋白激酶，是重要的信號傳導途徑之一，廣泛參與許多生命過程，包括生長、分化、腫瘤發生、細胞凋亡、細胞周期調控等。以小鼠卵母細胞為實驗對象，給予pka激動劑camp 、 8 - br - camp或磷酸二酯酶抑制劑imbx （ isomethylbutylxanthine ）均可抑制小鼠卵母細胞的胚泡破裂及減數分裂成熟。
  16. Inhibition regulation of apoptosis by insulin - like growth factor

    1對細胞凋亡的抑制調控
  17. Modulation of gabaergic inhibition in gustatory zone of the nucleus of the solitary tract

    能抑制對味覺信息的調製作用
  18. We should pay attention to adjusting the relative trading expense of earnest money, restraining transitional speculating behavior, preventing large customer control futures market, inspiring middle and small customer to trade, avoiding the interest distribution violating the rule of justice, the service charge of futures trading is the ex - pense that is handed in by the two sides of trading to the futures exchange house or the manage company, after the complishment of the futures contract, namely, the expense which is used for buyi

    在對保證金進行制度性調整時，應注意對保證金的相對交易費用進行調節，抑制過渡投機行為，防止大戶操縱期一貨市場，鼓勵中小投資者交易，避免利益分配有悻于公平、公正原則。期貨交易手續費是指期貨合同成交后，買賣雙方分別向期貨交易所或經紀公司繳納的費用，也即是用於購買經紀公司或者交易所提供的交易服務、管理的費用。
  19. This paper gives us too ways ： one is to adopt a better modulation mechanism, s - cdma, to bate noise jamming. the other way is to implement automatic frequency agility under strong noise, based on the ranging and burst configuration mechanism of current docsis protocol, meanwhile, guaranteeing cmts ' s normal operating. this paper comprehensively analyzes the anti - jamming mechanism of docsis, then presents the specific implementation of the latter method

    如何降低噪聲對網路的危害，本論文指出了兩個方向，其中一個就是在調制體制上下工夫，採用s - cdma體制，通過改變調制方式來主動的抑制噪聲的影響；另一個方向就是基於現在協議提供的抗噪聲干擾的機制，充分挖掘測距機制和靈活的突發配置機制的抗干擾潛力，以不影響頭端設備的正常運行為前提，以它們為基礎實現能自動避開強噪聲干擾的頻率轉移。
  20. Regulatory suppressor t cell

    調節抑制t細胞
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