逆轉作用 的英文怎麼說
中文拼音 [nìzhuǎnzuòyòng]
逆轉作用
英文
oversteepening-
T active immunization could maintain its castration for a period, at the age of 11w, the development of combs and wattles were evidently slower in the immunized groups ( p < 0. 05 ), and the testes atrophied. meanwhile, both its serum t concentration and body weight were evidently lower than the untreated group. 2. the does of 0. 2mg / ml produced the best immunisation performance and promotion growth effecct, we could induce the dosage was the main factor in deciding the final effect, injection at w3 achieved the best result
11周齡時，各免疫組雞體重、血漿睪酮含量顯著（ p 0 . 05 ）低於對照組，睪丸及冠垂發育受到抑制，明顯發生萎縮，其中以0 . 2mg / ml劑量， 3周齡免疫抑制作用最為明顯；此後隨著日齡的增長，免疫抑制作用逐漸下降，到15周齡時，免疫抑制作用明顯逆轉，機體的生長發育恢復正常，免疫各組與對照組相比測定指標無顯著性差異（ p 0 . 05 ） ；至19周齡，各免疫組睪丸和冠垂生長發育、體重、血漿睪酮含量都顯著（ p 0 . 05 ）高於對照組，其中以0 . 2mg / ml劑量組， 9周齡免疫促生長作用最為明顯。Translocation from endocytic compartments to the cytosol is the essential and rate - limiting step in the intoxication process of most toxins such as ricin, diphtheria toxin, shiga toxin and pseudomonas exotoxin ( pe ). a number of these toxins are transported to trans - golgi network ( tgn ), and in many cases such transport to the tgn is required for the translocation and cytotoxicity. in deed, 5 % of the ricin endocytosed by cells has been shown to reach the tgn
蓖麻毒素進入細胞的機理不甚明了，一般認為是rtb先與細胞膜受體結合，主要經過受體介導的內吞作用進入吞噬體，然後沿著內體、高爾基體、內質網等逆向分泌途徑，有序地運輸到內質網，最後從內質網轉位進入細胞漿，在胞漿內攻擊核糖體，從而抑制蛋白質的合成，導致細胞死亡。It is interesting that pma plus calcium ionophore a23187 can inhibit pma - induced pta1 expression, and this effect ca n ' t be reversed by calcmeurin inhibiter fk506. pta1 mabs can inhibit ctl activation and differentiation in mixed lymphocyte culture system when added at the beginning of the culture but can induce platelet activation and aggregation in the fc dependent manner. in 1997, pta1 cdna was cloned from cdna library of tpa activated jurkat cells, which belongs to immunoglobulin superfamily ( igsf ) with two v - like domains of extracelluar region of pta1
Il - 2 、 tnf - 、 pma可以使t細胞pta1表達上調， tgf -可以下調pta1的表達，而pma加上鈣離子載體a23187可以顯著抑制pma的上調作用，且這種抑制作用不被calcineurin抑制劑fk506所逆轉， 1997年burns教授從pma活化的jurkat細胞cdna文庫中克隆了pta1cdna全長，證實pta1是一個新分子，屬于免疫球蛋白超家族，胞膜外區有兩個v樣結構域。In this part the direct neurotrophic effects of inosine were explored
值得注意的是，肌昔的這些作用在其濃度到達1000vm時被逆轉。Clinical observation on reversal effect of weiansan on precancerous lesion of stomach carcinoma
胃安散逆轉胃癌前病變作用的臨床觀察Mdr1 express product p - glycoprotein was detected by immunocytochemical method and flowcytometry. the cytotoxicity and multidrug resistance reversion effect of tea polyphenol was examined by mtt assay in mcf - 7 and mcf - 7 adr carcinoma cell lines, and compared with pgp inhibitor quinidine. the pgp expression of mcf - 7 adr was strongly positive, the positive rate was 15 % ; the pgp expression of mcf - 7 was negative, the positive rate was 1. 8 %. ic50 of tea polyphenol to mcf - 7 and mcf - 7 adr is 115. 2g ml and 207. 6g ml respectively. ic50 of quinidine to mcf - 7 and mcf - 7 adr is 129. 8mol l 42. 1g ml and 94. 1mol l 30. 5g ml respectively. tea polyphenol and quinidine changed little toxicity of adriamycin to mcf - 7, but tea polyphenol and quinidine improved the sensitivity of mcf - 7adr to adriamycin significantly. immunocytochemistry and flow cytometry can detect p - glycoprotein expression level qualitatively and quantitatively. tea polyphenol is not only an anti - tumor agent, but also a multidrug resistant modulator similar as quinidine. tea polyphenol is advantageous for its little toxicity in tumor treatment
用免疫組化法和流式細胞儀對腫瘤細胞系mcf - 7和mcf - 7 adr的p -糖蛋白表達水平進行定性定量研究。用噻唑藍比色法mtt研究茶多酚的細胞毒性及其對耐藥性的逆轉作用，並與pgp抑制劑奎尼定進行了比較。免疫組化法檢測p -糖蛋白表達水平， mcf - 7 adr呈強陽性，而mcf - 7呈陰性流式細胞儀定量檢測結果mcf - 7 adr細胞系細胞陽性率為15 % ， mcf - 7細胞系細胞陽性率為1 . 8 % 。Exocytosis is the reverse of endocytosis.
出胞作用是內容作用的逆轉。The equations of motion governing the axisymmetric elastic deformation of finite orthotropic cylindrical shells, involving the effect of transverse shear and rotational inertia, are derived. by applying the reverberation method, the displacement and the resultant forces of the shell in the phase space are expressed. then the transient waves in the finite orthotropic cylindrical shell subjected to the axisymmetric impact are obtained by using inverse laplace transforms. furthermore, the transient solutions are decomposed to the generalized ray integrals and computed numerically
分析了計及剪切變形和轉動慣性的有限長正交異性圓柱殼中彈性瞬態波的傳播問題，採用回傳矩陣法，在相空間中給出了位移和內力的表達式。再利用laplace逆變換，得到正交異性圓柱殼受軸對稱沖擊作用時彈性瞬態波解，然後將其分解為若干廣義射線積分之和，並用數值方法求解之。Focused on " understanding media ", the article analyses in chapter i the key media theory of employing what could be of mcluhan in a thoughtful angle ， and advances the viewpoint of " human being - the extending of media ? ". in chapter ii 、 in the following third part ， the idea of " media ' s endding " is expounded and demonstrated ， which is composed of three parts ： extreme of speed ， content of the content ， and human being ' s last extending - the extending of central nerves 。 in the final part ， the general conclusion ? ? electronic media ' s influence on human behavior is given ， that is human are under universal narcissism and pacing to the bottom of it impelled by electronic speed. the article has five original points in it ： first of all, in virtue of " content theory " ， it analyzes the theory of " meida equals to information " by mcluhan from a scale theory stand. secondly, the tremendous power of electronic media is emphasized from an implosion view. in the third point, the article advanced originally that, after extension of central nerve system, human themsevels reverse to an extension of media and turns into a node of it ; at the same time, the article proposes that, all extensions before that of central nerve are " controllable extension ", however, it reverses to " uncontrollable extension " while extending ; then the authur put forward for the first time that, the concernful reason obsessing human is the failure of their in - body central nerve system " s jointing and controlling on out - body central nerve system naturally. 。 the fourth point is, the article demonstrates systematically the idea of " media ' s endding " from three levels solely by thoughtful means, and this is the most revolutionary point of it all
論文的創新之處有五點：一是從「尺度論」的角度，藉助「內容論」來解讀麥克盧漢的「媒介即訊息」的思想；二是從「內爆」的角度突出電子媒介的巨大力量；三是獨創提出在電子媒介時代，人類延伸了自己的中樞神經系統之後，人類本身被逆轉為媒介的延伸，成為電子媒介的一個節點；同時在本章節中提出中樞神經延伸之前的所有延伸為「可控延伸」 ，而中樞神經被延伸時「可控延伸」逆轉為「不可控延伸」 ，並首次提出，困擾人類種種問題的重要原因是體內的中樞神經系統無法無縫地對接和控制被延伸出去的體外中樞神經系統；四是獨自運用思辯的力量從三個層面綜合論證了「媒介的終結」的觀點，此處論證為本文最大創新之處，而此結論也將具有重大的理論與現實意義；五是根據以上幾個部分的論述，對人類目前的總體狀態作了一個定性的判斷，即：我們這個時代的迷惘在於我們深陷於自戀的漩渦里難以自拔，在電子媒介以其固有的電力速度的推動下，我們走向自戀的盡頭。Total cellular rna were extracted, 40 micrograms of the total rna were used in the reverse transcription reaction, using superscript ii reverse transcriptase, oligo ( dt ) i8 primers, and cy3 - dctp or cy5 - dctp for the experimental and control group respectively. the labeled cdnas were hybridized to microarrays at 42c for 12 h - 18 h
提取as _ 2o _ 3作用k562細胞前後的總rna ，用superscript逆轉錄酶逆轉錄成cdna第一鏈，並在逆轉錄的過程中，用cy3 cy5熒光染料分別標記對照組處理組，與自製的k562細胞基因表達譜晶元雜交。To follow the differentiation of lepcs in vivo, lepcs were tagged with reported gene egfp, dsred and lac z by retroviral methods
採用逆轉錄病毒介導的基因轉染手段，將lepcs標記lacz 、 egf戶和osreo作為報告基因。A c a2 + / calmo dul in - dep endent pro te in kinas e i i ( c amkii ) antagoni st kn - 6 2 ( 5x l0 - ' mol / l ) presented in the intemal solution had no significan effect on the current peaks induced by extracellular nmda ( l0 # mol / l ), but prevented the inhibitory effect of b on inmda " these results indicate that gcs have rapid, reversible idebitory effects on lnmda intracellular application of b thiough microelectrode had no effect on inmda, howevet, extracellular application of b or b - bsa suppressed peaks of inmda : all these denote b exerts its influence on nmda receptor by cytoplasm membrane mechansms, which is naxnely rapid, nongenomic mechhasms
加有快速、可逆的。非濃度依賴性抑制作用。通過微電極將b直接導入細胞內， inmda不受影響，而胞外給予b或besa ， inmda減小，提示b對nmda受體的調控通過膜機制產生，這種作用與經典的基因組機制不同；因為廣譜激酶抑制劑和特異的pka抑制劑均可翻轉b的抑制效應， camk11抑制劑也可阻斷b的效應，而這些激酶抑制劑本身對inmda即有強烈的抑制作用： pka激動劑本身對inmda無明顯作用，也不影響b對l 。Reverse malignant phenotypes of colonic carcinoma cells transfected with antisense gene to htrt
反義人端粒酶催化亞單位基因轉染對人結腸癌細胞惡性表型的逆轉作用Molecular mechanism and switching of tumor escape from the attack of
細胞攻擊的分子機制及逆轉作用Reversing effect of verapamil on multidrug resistance in human leukemia k562 cells
562細胞耐藥性的逆轉作用Reversal effect of artesunate on the multidrug - resistance of human leukemia k562 a02 cells
02細胞多藥耐藥的逆轉作用Tumor multidrug - resistance and reversing actions of traditional chinese medicine
腫瘤細胞多藥耐藥性與中藥的逆轉作用Study of tea polyphenol on the reverse of carcinoma cell lines multidrug resistance
茶多酚對腫瘤細胞多藥耐藥性逆轉作用的研究Effect of cyclosporine a, raloxifene and their combination on the reversion of multidrug resistace of k562 a02 line
干擾素對白血病細胞多藥耐藥的逆轉作用Effect of combined pioglitazone and fosinopril on reversing left ventricular hypertrophy in type 2 diabetes mellitus patients with hypertension
吡格列酮聯合福辛普利對2型糖尿病伴高血壓患者左心室肥厚的逆轉作用分享友人