高鉀血 的英文怎麼說
中文拼音 [gāojiǎxiě]
高鉀血
英文
hyperkalemia-
Ventricular fibrillation can occur in severe hyperkalemia.
在嚴重高血鉀時可以出現心室纖顫。Owing to persistent electrolyte abnormalities and renal salt loss, hormone studies were done and revealed elevated concentrations of cortisol, adrenocorticotropic hormone, aldosterone, renin and 17 - hydroxyprogesterone
但實驗室檢查卻發現持續性代謝性酸中毒，電解質失調包括低血鈉，高血鉀，低血氯和大量鈉離子由尿液中流失。Electrolyte analysis was consistent with acute tumour lysis syndrome ( atls ) with hyperkalaemia, hyperphosphataemia, hyperuricaemia, hypocalcaemia and elevated serum creatinine and lactate dehydrogenase levels
電解質分析結果符合急性腫瘤細胞溶解綜合征合併高鉀血癥、高磷血癥、低鈣血癥、血肌酐和乳酸脫氫酶水平升高。Intravenous infusions of concentrated potassium chloride corrects hypokalemia in patients underwent extracorporeal circulatin
體外循環術后低鉀血癥的高濃度靜脈補鉀治療The surviving time upon heat exposure and the swimming time were examined and the physiologic and biochemical changes such as body weight, potassium concentration, blood sugar, hemoglobin were compared before and after heat exposure
並比較了高溫適應前後以及高溫處理前後小鼠的一些生理生化指標如體重、血清中鉀離子濃度、血糖含量和血紅蛋白含量。After pretreated with nifedipine to block the influx of extracellular calcium, the vessels did not contract when they were stimulated by 60 mmol / l kc1 solution ; on the other hand, after pretreated with thapsigargin to deplete the intracellular calcium stores, the vessel remained the ability to contract when stimulated by 60 mmol / l kcl solution
使用ifedipine預處理血管，阻斷細胞外鈣離子內流，將血管置於含60mmol lkci的高鉀環境中，血管不發生收縮。使用細胞內鈣耗竭劑thapisin呷in預處理血管，耗竭細胞肌漿網內儲存的鈣離子。It is chloride - dependent and all the abnormalities can be corrected by thiazide diuretics which is the specific antagonist of the na - cl cotransporter ( ncct ). all these indicate that the disease should be relative to the ion transportation
它以高血壓，高血鉀和代謝性酸中毒為主要臨床表現，它是氯依賴性的，臨床異常能夠被鈉-氯共轉子ncct的特異性拮抗劑? ?雙氫克尿噻糾正，說明此疾病的發生和離子轉運相關。Effects of anion channel blocker on contraction induced by kc1 ( 60 mmol / l ) were observed
觀察陰離子通道阻斷劑對高鉀引起的血管收縮的舒張作用。The anion channel blockers nfa and nppb abolished the kcl - induced contraction of vessels which were pretreated by thapsigargin
將血管處于含60nunol lkci的高鉀環境中，血管發生收縮。而陰離子通道阻斷劑nfa 、 nppb可以抑制此收縮。Management of the hyperkalemia during cardiopulmonary bypass
體外循環中高血鉀不同處理方法研究Previous studies have showed that sulfonylureas possess a bidirectional effect on cardiovascular system : they can raise the coronary artery tone, aggravate ischemic injury to the cardiovascular tissues, and accelerate cardiac hypertrophy through closing cardiovascular atp - sensitive k ( superscript - ) channels ; meanwhile they can also regulate glucolipid metabolism, modulate inflammatory reaction and inhibit atherosclerosis through activating peroxisome proliferators - activated receptors
摘要基礎研究表明，磺脲類藥物對心血管系統有雙向影響：磺脲類藥物能夠關閉心血管系統atp敏感性鉀通道，從而增高冠脈張力，加重缺血對心血管組織的損傷和促進心臟肥厚的發生；另一方面，磺脲類藥物能夠激活過氧化物酶體增殖物激活受體，從而調控糖脂代謝，調節炎癥反應和抑制動脈粥樣硬化的發生。Results the frequency of discomfort, low blood pressure and arrhythmia in the treatment group is lower than that in control group ( p < 0. 01 ) but the density of kalemia in serum does not obvious change
結果高鉀透析組患者在不適癥狀、低血壓、心律失常發生率等方面均顯著低於對照組（ p < 0 . 01 ） ，而透析后血清鉀濃度無明顯改變。It has been demonstrated that kvl. 2, an isoform of delayed - rectifier potassium channel, is up - regulated in the ischemic neurons. besides, the activity of kvl. 2 is tyrosine phosphorylation dependent, phosphorylation of the tyrosine residue in the amino terminus of the channel by tyrosine kinases suppresses the channel current. therefore, we hypothesize that vegf could enhance the phosphorylation of kvl. 2, which results in an inhibition of / k
在介導i _ k的鉀通道家族中， kv1 . 2是其中的一個亞型，研究報道它在缺血神經元中的表達是增高的；此外，它的活性與其酪氨酸磷酸化水平相關，在其氨基末端的酪氨酸殘基經酪氨酸激酶磷酸化后可顯著降低其介導的電流的幅度。Therefore, adopting a low sodium, high potassium, high magnesium and high calcium diet is a prudent strategy for the prevention and treatment of high blood pressure
因此，食用低鈉高鉀高鎂和高鈣的膳食是預防和治療高血壓的明智策略。分享友人