pkc 中文意思是什麼

pkc 解釋
公開密鑰密碼體制
  1. On cytomembrane ; resolved the precipitation in buffer d ( contained triton x - 100 ) shattered by ultrasound, 4 vibrated for 60min, centrifuged at 45000g for 1 hour, took the supernatent to mesure protein concentration and then mesure the pkc activity

    目前，在鉛的神經毒性研究過程中，鉛對pkc的作用引起人們極大關注。這是因為鉛比鈣對pkc有更高的親和力。現已知， ph卜可以取代caz 」激活蛋白激酶c 。
  2. The activity of pkc on cytomembrane in mice brain showed ; 1 ) the value of pkc activity was lower than that of cytoplasma, 2 ) the activity of each group reached the peak at p22, 3 ) the value of pkc activity varied with lead concentration, especially for the 9. 6mmol / l group

    2 ）鉛暴露小鼠腦細胞膜pkc活性總體上比胞漿低， pkc活性高峰時段在p22左右，胞膜pkc活性隨鉛濃度的升高而升高。其中9 6nunoul組升高最為明顯。
  3. Bovine sperm extract or sr2 + - induced ca2 + oscillations in mii oocytes and distribution of protein kinase c a ( pkc a ) in both mil oocytes and early embryos were not differed between young and old mice. electrofusion rates were equivalent among 9 types of gv - cytoplast complexes constructed by gv exchange between 6 - 8 - week - old mice and 6 - 8 - week - old, 3 - month - old, 6 - month - old, 9 - month - old and 12 - month - old mice respectively

    6 - 8周齡小鼠gv期卵母細胞分別與6 - 8周齡、 3月齡、 6月齡、 9月齡和12月齡小鼠gv期卵母細胞進行gv互換所形成的9種gv -胞質體復合體的融合率和9種重組卵母細胞的成熟率並不因小鼠年齡的增長而有所改變。
  4. They are pkc, pka, mek1 in erk pathway, mekk1 in sapk / jnk, p38 in p38 pathway, the three latter pathways all belong to the map kiiiase pathway. the inhibition of pkc decreases the proliferation after the il - 16 treatments, which hint its role in proliferation signal transduction

    結果表明，加入pkc的抑制劑，強烈抑制了所有濃度16處理下細胞的增殖，暗示pkc在u一16與cd4結合后，可能傳遞了增殖相關信號，它的存在是jllrk盯細胞的生長增殖必須的。
  5. Measured the protein content by lowery method and the activity of pkc by advanced takai method

    蛋白激酶c是一種鈣活化的、磷脂依賴性的絲氨酸蘇氨酸激酶。
  6. After breast feeding, their descendents were fed with the same water, then killed them on the 1st, 8th, 15th, 22nd, 30th day after birth respectively and took out of the brains, stored at - 70. ( 2 ) measurement of the activity of pkc in brains : in cytoplasma ; took out the samples, cut the hippocampi into pieces and added buffer ( glucose 0. 32mol / l, hepes ph 7. 5 ), then shattered with ultrasound, centri - fuged at 27000g for ih at 4ti, moved the supernant into a new tube

    本研究以細胞信號轉導途徑為線索，分別探討了pkc 、 erk 、 nos1等信號分子及即早基因（ c - fos ， c - jun ）在慢性鉛暴露小鼠腦細胞中的活性、蛋白表達及mrna表達的改變，以及鉛暴露小鼠記憶行為的變化，並將細胞中信號分子的改變與記憶功能改變結合進行分析以尋找一些內在規律。
  7. Under normal culture conditions, pkc - dependent repulsion dominated, but the repulsion could be converted to attraction by inhibiting pkc or by elevating cytosolic cgmp

    通常條件下， pkc通路介導了生長錐排斥反應，而ip3通路介導了吸引反應，由於pkc通路占據主導， sdf - 1和baclofen排斥生長錐。
  8. This response was mediated by gi and subsequent activation of phospholipase c ( plc ), which triggered two pathways : protein kinase c ( pkc ) led to repulsion, and inositol 1, 4, 5 - triphosphate ( ip3 ) receptor activation led to attractive turning

    信號轉導機制的研究表明， sdf - 1和baclofen激活了生長錐表面的g蛋白耦聯受體，通過gi將信號傳遞給plc ，進而激活pkc和ip3信號通路。
  9. In order to find an explanation and to determine the possible mechanism of these biological effects, many in vitro and in vivo studies of emf effects have been published. these studies were involved in many enzymes ( such as odc, succinate dehydrogenase, pkc ), pro - oncogene ( c - myc, c - fos, etc. ), gap junction and protein synthesis, and so on

    為了揭示工頻磁場生物效應的機制，尤其是與腫瘤發生發展的關系，人們開展了較為廣泛的體內及體外的實驗研究，涉及到各種生物酶類（ odc 、琥珀酸脫氫酶、 pkc等） 、原癌基因（ c - myc 、 c - fos等） 、細胞間縫隙連接及蛋白合成等方面。
  10. There are still many questions remain unanswered, including the mechanisms that specify oligochitosan recognition, the participation of additional molecules in transmembrane signaling, and the repertoire of downstream pathways activated by oligochitosan. it was also observed that pdtc did not totally block no production, which gave us another evidence that there must be some other signal transduction pathways ( perhaps mapk, pkc, ca2 + dependent signaling ) involved in the activation, so further investigation is necessary to clarify molecular mechanism of oligochitosan inducing immunological en

    Westemblot結果顯示100林歲ml劑量的殼寡糖作用於raw264 . 7細胞（ 4x105個細胞/ ml ） 4h ，胞核內p65的含量明顯增加， 6h時達到高峰， 10h時恢復到基礎表達量，這就提示在殼寡糖對巨噬細胞的激活過程中nf一kb活化狀態可以持續6個小時。
  11. We just want to study and analyze the effects of different concentration of lead on the activity of pkc and the function of learning and memory

    鉛具有很強的神經親和性，可在神經組織中蓄積，引起神經系統功能（主要是學習記憶功能）的長期損害。
  12. Introduction protein kinase c ( pkc ) is a number of serine / threonine kinase family which play central regulatory roles in a multitude of cellular processes, including cell proliferation, cell cycle progression, differentiation, apoptosis. increasing evidence from studies using in vitro and in vivo systems suggests that pkc is a key regulator of critical cell cycle transitions, including cell cycle entry and exit and the gl and g2 checkpoint

    前言細胞周期是細胞增殖、分化的一個重要過程，它的啟動與進程受到嚴格調控，參與調控的因子不僅包含起正調控作用的細胞周期素（ cyclins ） 、細胞周期蛋白依賴性激酶（ cyclin - dependentkinase ， cdk ） 、還有起負調控作用的cdk抑制因子（ cyclin - dependentkinaseinhibitor ， cki ） 。
  13. 5 mmol / l and 2. 4 mmol / l group reached the peak at p30. after p15, the activity of pkc increased when treated with low concentration of lead but decreased with high concentration of lead

    4mmoul鉛暴露組的pkc比活性在至p22則不但未見降低，反而繼續升高m2至ppo又繼續升高，成為最高點。
  14. Results ( 1 ) effect of different concentration of lead on the activity of pkc for the control, the activity of pkc increased gradually at the early devel - opping stage and rose sharply on the 15th day, reached the peak, then decreased on the 22nd day and increased shortly until the 30th day with the activity almost turned back to the level at birth or even lower. compared with the control, groups treated with different concentration of lead showed a graph of different cytoplasmic activity of pkc. most of the groups reached the peak at p15, but the 0

    L ）鉛暴露小鼠腦胞漿pkc活性與正常組相比可見一定的差異，多數組第15日問）為最高峰，低濃度染鉛組在某些時段中pkc比活性高於正常組，而隨著鉛濃度的升高，這些時段的pkc活性逐漸降低，較高鉛濃度組酶活性均低於正常組； inunovl汐）組的p22以後，比活性迅速升高，至p30時比對照組高出很多。
  15. Pkc, an enzyme that is activated by the receptor - mediated hydrolysis of inositol phospholipids, can lead to the increase or disturbance of the transcription of some genes when stimulated persistently

    Pkc是一類ca入活化，磷脂依賴性蛋白激酶，當其處于持續激活狀態時會導致某些基因轉錄活性的增強、代謝紊亂。
  16. Our previous work has show that sh2a may relate to tumor onset or development, and our current research suggests that sh2a gene participate tumor onset or development by altering the pkc activity in cytoplasm

    前期研究已表明， sh基因表達與腫瘤發生發展有一定相關性，通過本實驗，我們認為，可能是通過sh 。 a蛋白對胞漿蛋白中pkc活性改變，參與腫瘤的發病過程。
  17. In both cell lines ex - pressing sh2a gene, the cytoplasm pkc activity decreased 40 %, but no apparent alteration was found in mapk, tpk and membrane pkc activity. the proliferation index of transfected cells exhibited an decline in cytometry detection

    L sh的肝癌bel7402細胞及cos7細胞胞漿蛋白中的pkc活性均下降達40 ，胞膜pkc 、胞漿mapk ， 』 fpk活性未見明顯改變，轉染細胞經流式細胞儀檢測增殖指數有所下降。
  18. We have obtained amount of expression proteins through the gene recombinant technique and detected its effect on the activity of pkc, which is crucial to further study

    通過基因重組技術大量獲得表達蛋白，並檢測了其對蛋白激酶c （ pkc ）活性的影響，對進一步研究新蛋白的生物學功能具有重要的意義。
  19. The term protein kinase c ( pkc ) stands for a group of at least twelve serine / threconine kinases that are characterized by a high degree of similarity in their catalytic kinase domains and cysteine - rich regions

    前言蛋白激酶c （ pkc ）是一組絲蘇氨酸蛋白激酶，參與多種信號傳導系統。包括細胞分化、生長調控、腫瘤發生和細胞凋亡。
  20. The oocytes reconstructed by gv exchange between young and young or young and aged mice showed normal bovine sperm extract or sr2 + - induced ca2 + oscillations in mii stage and normal pkc a distribution in mii oocytes and early embryos. normal meiosis appeared in the oocytes reconstructed by gv exchange between young and young mice

    在年輕小鼠卵母細胞之間以及年輕小鼠與老齡小鼠卵母細胞之間gv互換后的重組卵母細胞表現出正常的牛精子抽提物或sr ~ （ 2 + ）誘發的ca ~ （ 2 + ）振蕩，以及在減數分裂成熟和早期胚胎中表現出正常的pkc分佈。
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