胞脈不通 的英文怎麼說
中文拼音 [bāomàibùtōng]
胞脈不通
英文
vascular obstruction of uterine collaterals-
Not only was the pattern of recordings along the artificial lateral line similar to the pattern recorded from the nerve cells of a real fish, but the computer could also decipher it to follow the source of the pulse as it moved along different paths
結果顯示不僅該系統記錄的圖形與從真正的魚身上的神經細胞所記錄的圖形相似,而且配套的電腦程序還能通過對記錄圖形的解讀來追蹤沿著不同路徑移動的脈沖水波的源頭。The essentially universal biophysical phenomenon of " electroporation " occurs if an appropriate pulse field is applied. electroporation is believed to be the rapid creation of aqueous pathways through lipid - containing barriers in cells and tissue. the driving force is the physical interaction of electric fields with different dielectric constants
電穿孔效應是指在適當高壓脈沖電場作用下,細胞或組織間起相對隔離作用的「屏障」內快速形成液態通道的現象,是電場與具有不同介電常數而且易變形的物質相互作用的結果。Part iii hvec, vsmc and fibroblast were seeded onto inner surface of tubular porous pga and phb scaffold precoated with collagen. sead hvec, vsmc and fibroblast onto three porous polyglycolic acid and poly - ? - ydroxybutyrate scaffolds precoating with collagen, which different caliber but embedded each other. observe cell growth and differentiation on these scaffolds
通過用膠原分別包埋處理的三個管徑大小不同、但能相互嵌套的pga及phb管形支架,並種植人臍靜脈內皮細胞、人血管平滑肌細胞、人成纖維細胞進行三維立體生長培養,觀察細胞生長分化情況。To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation
為了驗證上述假設,本文分別在整體和離體水平,採用大腦中動脈缺血( middlecerebralarteryocclusion , mcao )和體外氧?糖剝奪( hypoxiaandglucose - deprivation , hgd )缺血模型,首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況,觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化,然後通過外源性給予vegf蛋白,在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響,最後利用vegf反義脫氧寡核苷酸( oligodeoxynucleotide , odn )特異阻斷內源性vegf蛋白的表達,觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用,以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。分享友人