腫瘤生長因子 的英文怎麼說

中文拼音 [zhǒngliúshēngzhǎngyīnzi]
腫瘤生長因子 英文
tumor growth factor
  • 腫 : Ⅰ名詞（隆起處） swelling Ⅱ動詞（突起） swell; be swollen
  • 瘤 : 名詞（瘤子） tumour
  • 生 : Ⅰ動詞1 （生育; 生殖） give birth to; bear 2 （出生） be born 3 （生長） grow 4 （生存; 活） live;...
  • 長 : 長Ⅰ形容詞1 （年紀較大） older; elder; senior 2 （排行最大） eldest; oldest Ⅱ名詞（領導人） chief;...
  • 因 : Ⅰ動詞[書面語] （沿襲） follow; carry on Ⅱ介詞1 [書面語] （憑借; 根據） on the basis of; in accord...
  • 子 : 子Ⅰ名詞1 （兒子） son 2 （人的通稱） person 3 （古代特指有學問的男人） ancient title of respect f...
  • 腫瘤 : tumour; core （綿羊體內的）; neoplasm; phyma
  • 生長 : grow; grow up; ascent; merisis; build up; auxesis; increment; overgrowth; gain; burgeon; bourgeon...
  1. Comprehensive cellular responses was found in human amnion fl cells following exposure to low concentration of mnng, such as the lowering of dna replication fidelity resulted from alteration of dna polymerase profile ; activation of a lot of transcription factors, such as api, creb, nf - kb etc ; clustering of egfr ( epidermal growth factor receptor ) and tnfr ( tumor necrosis factor receptor ) and activation of camp - pka - creb and jnk / sapk signal pathways

    我們發現，低劑量mnng處理后的人羊膜fl細胞有廣泛的細胞反應，並有多個信號轉導通路的激活和基因表達的改變。例如dna復制保真度下降， dna聚合酶譜發生改變，應用報告基因技術和底物磷酸化檢出技術證明細胞一系列轉錄因子如ap1 、 creb 、 nf b等被激活，細胞表面受體如表皮生長因子受體、腫瘤壞死因子受體發生聚簇，細胞信號轉導通路camp - pka - creb和jnk sapk被激活。
  2. The b lymphocyte stim lator ( blys ) also known as baff, tall - 1, thank, ztnf4, is the most recent addition to the tumor necrosis factor family ( tnf ) ligands. blys induces both in vivo and in vitro b cell proliferation, differentiation and immunoglob lin secretion. meanwhile, it also strongly suppresses the growth of tumor cell lines. the over - production of blys is associated with the development of certain autoimmune disease, such as systemic lupus erythaematosus ( sle ), rheumatoid arthritis, myasthenia gravis, sj greh ' s syndrome

    人b淋巴細胞刺激因子（ hblys ）是1999年發現的腫瘤壞死因子超家族成員。最早發現其可誘導腫瘤細胞凋亡，之後發現它是一個強有力的b細胞共刺激因子，它在體外及轉基因動物體內可明顯刺激b細胞的生長，免疫球蛋白分泌，並導致動物自身免疫性改變。
  3. For example, we have found the clustering of egfr ( epidermic growth factor receptor ) and tnfr ( tumor necrosis factor receptor ) and the activation of camp - pka - creb and jnk / sapk pathways after mnng treatment

    例如細胞表面受體如表皮生長因子受體、腫瘤壞死因子受體發生聚簇，細胞信號轉導通路camp kacgyb和jnk sapk被激活。
  4. Platelets and many kinds of their adhesins play an important role in every step of tumors growth and metastasis, therefore new methods of tumor - related treatment could be found aiming at platelets or their adhesins, which would be amazing if they are applied to the clinical field

    血小板及其多種粘附分子在腫瘤生長和轉移的各階段都發揮了重要作用，其中任何一個因素都可能成為腫瘤治療的新靶點，這就為臨床腫瘤治療提供了廣闊的思路和豐富的手段。
  5. Vascular endothelial growth factor and the effects of endostatin on the inhibition of tumor angiogenesis in treating hepatocarcinomas

    血管內皮生長因子和內皮抑素在肝癌抗腫瘤相關血管治療中的作用
  6. Dendritic cells ( dc ) is the most powerful apc, which can markedly increase the antigen - presentation capacity by maximizing the pepitide - mhc complexes on the cell surface and upregulating the co - stimulatory ligands b7 - 1 and b7 - 2, adhesion moleculees such as il - 12 that promote full activation of lymphocytes. full activation of antigen - specific t cells requires two signals - one signal coming via the tcr and the other signal through engagment of co - stimulatary molecules. t cells receiving one signal via their tcr are turned off by mhc ( major histocompatibility complex ), via t cell cd28 binding to b7 on the dc induce tlymphokine and t cell proliferatiion

    T細胞介導的細胞免疫在控制腫瘤生長方面發揮著重要作用， t細胞在發揮抗瘤效應（分泌細胞因子和直接殺傷）之前必須先經過活化，體內專職抗原提呈細胞（ apc ）細胞並使其活化，樹突狀細胞（ dendriticcell ， dc ）為t細胞的激活提供雙重信號， t細胞藉助tcr識別由dcmhc分子遞交的抗原肽后，通過tcr - cd3復合體傳遞抗原特異性識別信號（第一信號） ，以cd28為主的t細胞表面輔佐分子識別dc表面b7分子，傳遞非特異性協同刺激信號（第二信號） ，在機體抗腫瘤免疫應答中處于核心地位。
  7. Indeed, researchers led by jain have shown that antiangiogenic factors can “ normalize ” tumor vasculature before killing it by pruning excess, inefficient vessels while leaving efficient vessels temporarily intact

    的確，由詹恩所領導的團隊發現：抗血管新生因子在殺死腫瘤之前，會修剪過度生長、沒有效率的血管，並讓有效率的血管暫保無恙，而使腫瘤的血管系統正常化。
  8. Advance in tumor angiogenesis inhibitors targeting vegf and its receptors

    以血管內皮生長因子及其受體為靶點的腫瘤血管生成抑制劑的研究進展
  9. Sleep / waking cycle is a complex network modulation and many factors such as interleukin - 1 ( il - 1 ), tumor necrosis factor ( tnf ), growth hormone releasing hormone ( ghrh ), vasoactive intestina polypeptide ( vip ) and many conventional neurotransmitters such as serotonin ( 5 - ht ), acetylcholine ( ach ), norepinephrine ( ne ), dopamine ( da ) and gamma - aminobutyric acid ( gaba ) were involved in it. recent evidence has shown that no synthesized in neurons in several areas of the brain can induce the release of neurotransmitters. in the rat central nervous system, the anatomical distribution of nos - containing neurons is now well established, and it was reported that nos is co - localized with neurotransmitters well known for their involvement in sleep mechanisms, i. e. 5 - ht, ach, da and gaba

    鄭州大學2003屆碩士畢業論文gaba受體激動劑消除no合成酶抑制劑對大鼠睡/醒周期的影響睡/醒周期的形成是一個復雜的網路調控的結果，體內許多因子都參與了這一調控網路，這些因子如白介素一1 （ il一1 ） 、腫瘤壞死因子（ tnf ） 、生長激素釋放激素（ ghrh ） 、血管活性腸膚（ vip ）以及經典的神經遞質如5一輕色氨（ 5一ht ） 、乙酞膽堿（ ach ） 、去甲腎上腺素（ ne ） 、多巴胺（ da ）和卜氨基丁酸（ gaba ）等，它們在睡眠的發生和調節中也發揮著重要作用。
  10. 2 ) basis of upon studies, we have also designed and sythesized the mutation ii of the cmiv that been greatly changed in the 3 " of the gene comparing with nature cmiv : the ht gf3 ( the third loop region of htgf2 specifically binding to egfr receptor ) was fused to 3 " of gene of cmiv through a flexible linker. the gene of the mutation ii of cmiv was sequenced and clonged to the vector of ptxfus to fuse to the 3 " of gene of thioredoxin

    二、在以上研究的基礎上，對cmiv抗菌肽的c端進行較大的改造，即將與腫瘤細胞過度表達的表皮生長因子受體（ egfr ）具有高親和性的因子多肽tgf _ 3通過疏水柔性接頭連接在抗菌肽cmiv的c端，設計完整的基因，並在大腸桿菌中利用ptxfus表達載體與硫氧還蛋白進行可溶性融合蛋白表達。
  11. Many types of tumour produce far more egfr than normal cells do, and one effect of that is to stimulate the proliferation of tumour cells

    許多種類的腫瘤可產生比普通細胞高得多的表皮生長因子受體，影響之一就是刺激了腫瘤細胞的擴散。
  12. Discussions angiogenesis, an essential process for tumor growth, is regulated by endothelial proliferation factors and their inhibitors such as angiostatin

    討論血管生成對于腫瘤生長來說是一個必需的過程，受內皮細胞增殖刺激因子和抑制因子的共同調控。
  13. In summary, we used the cdna microarrays to analyze the differentially expressed genes in the arsenic trioxide induced k562 cells. our results further confirmed in gene expression level that growth arrest and apoptosis could be induced by arsenic trioxide. such induced apoptosis may also involve the functional inhibition of igf

    應用基因晶元技術研究aszo3作用前後k562細胞的基因表達變化，與以往的研究結果相一致，也是通過抑制細胞生長、誘導細胞凋亡而發揮抗腫瘤的作用，在這個過程中， aszo3可能對胰島素樣生長因子有抑制作用。
  14. Inhibition of tumor - induced angiogenesis may represent an effective cytostatic strategy. angiostatin inhibits endothelial proliferation, angiogenesis, and tumor growth., so it is very important to receive a great deal of soluble, correctly folded, biologically ac

    Angiostatin已被證實是一種很具潛力的抗腫瘤生長和轉移的因子，因而若獲得大量具有高活性、高純度的angiostatin對腫瘤的治療會有重要的意義。
  15. Objective cells will receive mutiple stimulation of external signals during growth and differentiation. among these signals, growth factors have important effects on cell life

    目的細胞在生長、分化過程中不斷的接受外界刺激，其中生長因子對細胞的生長、分化具有重要作用，生長因子與腫瘤的發生過程也有密切的關系。
  16. Epidermal growth factor receptor and its signal transduction pathway in tumor - targeting therapy with chinese and western medicines

    表皮生長因子受體及其信號轉導通路與中西藥腫瘤靶向治療
  17. We are already looking at using the same technique for colorectal cancer, as we beliee we could modify the bacteria to produce factors that will reduce tumor growth

    我們已經開始研究用同樣的技術來治療結腸直腸癌癥，因為我們相信我們可以改造出一種細菌產生抑制腫瘤生長的因子。
  18. When the body suffered from malignancy, on the one hand, tumor restrained cell immunity function, antibody ' s production and the body ' s immunity effect through inducing restraining t cell, so it accelerated tumor ' s growth, on the other hand, tumor induced body fluid restrained gene, which prevented t cell and nk cell from killing tumor and restrained lymphocyte to mitotic multiplication reaction, indeed it accelerated tumor ' s growth

    當機體一旦患上惡性腫瘤后，一方面腫瘤通過誘導抑制性t細胞，抑制細胞免疫功能及抗體的產生，抑制機體對腫瘤的免疫效應，從而使腫瘤逃逸機體的免疫監視，促進腫瘤的生長。另一方面，腫瘤通過誘導體液抑制因子阻止t細胞和nk細胞的等對腫瘤的殺傷作用，抑制淋巴細胞對有絲分裂原的增殖反應，甚至促進腫瘤的生長。
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