acetylcholine (ach) 中文意思是什麼

acetylcholine (ach) 解釋
乙酰膽堿
  1. In order to investigate the relationship between myocardium responsiveness and its firing dynamic states, bilateral vagotomy, m receptor blocking, acetylcholine ( ach ) perfusion and continuously autonomic nerve ending stimulating methods were employed to change the rabbit sinoatrial node ( sa ) firing dynamics. isoprenaline, noradrenalin, ach, sympathetic or vagal nerve ending stimulation was used individually to measure the sa responsiveness in different dynamic state

    3 、竇房結在正常放電狀態時,對0衛, 0人0石ug kg異丙腎上腺素的反應性均強于應用阿托品后,前後相比有顯著差異( p 0刀5 ) ;待阿托品作用部分消除后,竇房結對異丙腎上腺素的反應性也部分恢復。
  2. To examine the effect of ach, the researchers looked at the brain ' s nicotinic acetylcholine receptors ( nachr ), to which ach binds to stimulate neural activity

    為了研究乙酰膽堿的作用,研究者們研究了大腦了的煙堿乙酰膽堿受體,煙堿乙酰膽堿受體是乙酰膽堿的結合部位,並刺激神經活動。
  3. Sleep / waking cycle is a complex network modulation and many factors such as interleukin - 1 ( il - 1 ), tumor necrosis factor ( tnf ), growth hormone releasing hormone ( ghrh ), vasoactive intestina polypeptide ( vip ) and many conventional neurotransmitters such as serotonin ( 5 - ht ), acetylcholine ( ach ), norepinephrine ( ne ), dopamine ( da ) and gamma - aminobutyric acid ( gaba ) were involved in it. recent evidence has shown that no synthesized in neurons in several areas of the brain can induce the release of neurotransmitters. in the rat central nervous system, the anatomical distribution of nos - containing neurons is now well established, and it was reported that nos is co - localized with neurotransmitters well known for their involvement in sleep mechanisms, i. e. 5 - ht, ach, da and gaba

    鄭州大學2003屆碩士畢業論文gaba受體激動劑消除no合成酶抑制劑對大鼠睡/醒周期的影響睡/醒周期的形成是一個復雜的網路調控的結果,體內許多因子都參與了這一調控網路,這些因子如白介素一1 ( il一1 ) 、腫瘤壞死因子( tnf ) 、生長激素釋放激素( ghrh ) 、血管活性腸膚( vip )以及經典的神經遞質如5一輕色氨( 5一ht ) 、乙酞膽堿( ach ) 、去甲腎上腺素( ne ) 、多巴胺( da )和卜氨基丁酸( gaba )等,它們在睡眠的發生和調節中也發揮著重要作用。
  4. In 1980, furchgott and his colleagues presented the first evidence that acetylcholine ( ach ), by acting on endothelial cells, stimulated the release of a relaxing factor ( edrf ) that acted on the preconstricted rabbit thoracic aorta to produce relaxation. a research interest on the endothelium - derived regulation of vascular tone, since then, is on the increase

    自1980年furchgott發現乙酰膽堿( acetylcholine , ach )可通過內皮釋放一種舒張因子( edrf ) ,使預先收縮的免主動脈環鬆弛以來,人們對內皮源性血管張力調節機制進行了廣泛的研究,現已證實, edrf便是一氧化氮( no ) 。
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