endogenous cell 中文意思是什麼

endogenous cell 解釋
內原細胞
  • endogenous : adj. 1. 【生物學】內生的，內長的，內原的。2. 【地質學；地理學】內成的。
  • cell : n 1 小室，單室；隔間，艙；〈詩〉茅舍；（單個的）蜂窩，蜂房。2 〈詩〉墓穴，墓。3 （大修道院附屬的...
  1. While the suppression of ( - arrestin2 endogenous expression by antisense or rnai technology considerably attenuated sdf - 1 ( - induced cell migration. expression of ( - arrestin2 also augmented chemokine receptor ccr5 - mediated but not egf receptor - mediated chemotaxis, indicating the specific effect of ( - arrestin2

    與此相似，另一種趨化因子受體ccr5介導的趨化作用也受- arrestin2的調控，但egf受體介導的趨化作用則不受- arrestin2的調控，說明- arrestin2的作用有一定的受體特異性。
  2. Background : ghrelin is a 28 - amino acid endogenous peptide recently identified in the secretory granules of x / a - like cells in the rat stomach, which can act on the growth hormone secretagogue receptor to accelerate the secretion of growth hormone. it was identified that ghrelin - immunoreactive cell and its receptor localize in areas of brain, such as hypothalamus ( pvn, arc ), hippocampus, adenohypophysis and so on. motilin is a 22 - amino acid peptide secreted from the upper part of the small intestine, which regulates the interdigestive motility of gastric

    背景資料： ghrelin是新發現的由28個氨基酸組成的內源性腦腸肽，由胃部泌酸腺x / a樣內分泌細胞分泌， ghrelin免疫活性神經元及其受體在中樞神經系統中如下丘腦室旁核（ paraventricularneucleus ， pvn ） 、弓狀核（ arcuatenucleus ， arc ） 、海馬區及腺垂體等均有不同濃度的分佈，可與生長素促泌物受體（ growthhormonesecretagoguereceptor ， ghsr ）結合促進生長素（ growthhormone ， gh ）分泌。
  3. Shengyuan maintenance blood - sugar capsule is chooses the cactus, the balsam pear, the black bee bee glue, the kudzu root four pelts the sugar ingredient reasonably to blend, purifies the highly effective multi - skill health product by the modern biotechnology extract which but becomes, has the bidirectional adjustment, the control blood sugar, the activation and the nutrition island of langerhans beta cell, the promotion endogenous secretions insulin secretion, enhances the human body sugar metabolism and the fat metabolism ability, strengthens the pancreas function the function, at the same time also has the repair diabetes liver, the kidney damage, the prevention and the improvement diabetes illness complication function

    聖源維糖軟膠囊是選擇仙人掌、苦瓜、黑蜂蜂膠、葛根四大降糖成份合理配伍，以現代生物科技萃取精製而成的高效復合型健康產品，具有雙向調節、控制血糖，激活和營養胰島細胞，促進內源性胰島素分泌，提高人體糖代謝和脂肪代謝能力，增強胰臟功能的作用，同時又具有修復糖尿病肝、腎損傷，預防和改善糖尿病並發癥的作用。
  4. Endogenous elicitor activity, termed cdw, was prepared from the cell walls of suspension - cultured ginseng ( panax ginseng c. a. meyer ) cells via cellulase degradation. cdw activated in vitro the nadph oxidase activity of isolated plasma membranes and stimulated in vivo h2o2 generation by ginseng cell suspensions

    在本研究工作中，金瓜炭疽細胞壁激發子與脫乙酰幾丁質是來源於真菌細胞壁的真激發子，人參細胞壁降解物與寡聚半乳糖醛酸是來源於植物細胞壁的內源激發子。
  5. We concluded that excessive expression of exogenous htr gene may compete with the endogenous telomerase rna and prevent rna template from combining with telomeric dna, thus repressing the elongation of telomeric dna ( telomeres ) and causing cell aging and cell death. - 6 - 3. some modifications have been made to overcome the limitation of conventional telomeric repeat amplification protocol ( trap ) assay

    分析其原因，可能是htr基因的過表達在數量和空間效應上同細胞內的端粒酶rna組分產生竟爭，一定程度上阻礙了端粒酶rna模板區與端粒dna的結合，從而抑制端粒dna的延伸，導致細胞凋亡。
  6. To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

    為了驗證上述假設，本文分別在整體和離體水平，採用大腦中動脈缺血（ middlecerebralarteryocclusion ， mcao ）和體外氧?糖剝奪（ hypoxiaandglucose - deprivation ， hgd ）缺血模型，首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況，觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化，然後通過外源性給予vegf蛋白，在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響，最後利用vegf反義脫氧寡核苷酸（ oligodeoxynucleotide ， odn ）特異阻斷內源性vegf蛋白的表達，觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用，以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。
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