proinflammatory 中文意思是什麼

proinflammatory 解釋
促炎癥反應
  1. No, a first gas information molecule discovered in human being, is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels. in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated. in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no, the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation, both of which are demonstrated to induce the

    在內毒素休克過程中vec是lps及其誘導機體產生的多種促炎細胞因子如tnf 、 il - 1作用的主要靶細胞, vec誘導型一氧化氮合酶( induciblenitricoxidesynthase , inos )激活、 no大量誘生而內皮型一氧化氮合酶( endothelialnitricoxidesynthase , enos )活性被抑制、 no生成障礙,是血管反應性異常變化、血管調節機制紊亂的重要發病環節。
  2. The major factors for progressie loss of ? - cell function and mass are glucotoxicity, lipotoxicity, proinflammatory cytokines, leptin, and islet cell amyloid

    導致細胞的功能與體積進行性損害的因素包括糖毒性、脂肪毒性、免疫炎性因子、瘦蛋白與胰島細胞澱粉樣變。
  3. The major factors for progressive loss of ? - cell function and mass are glucotoxicity, lipotoxicity, proinflammatory cytokines, leptin, and islet cell amyloid

    導致細胞的功能與體積進行性損害的因素包括糖毒性、脂肪毒性、免疫炎性因子、瘦蛋白與胰島細胞澱粉樣變。
  4. Objective : markers of neuroinflammation, including activated microglia and increased levels of circulating proinflammatory cytokines, have been observed in the brains and csf of patients with parkinson disease ( pd )

    目的:研究發現,在帕金森病( pd )患者的腦組織和腦脊液中存在炎癥反應,表現為活化的小膠質細胞和促炎細胞因子水平增高。
  5. Recent observations suggest that proinflammatory cytokines can modulate cardiovascular functions by a variety of mechanisms

    最近的研究提示炎性細胞因子可以通過多種不同的機制而調節心血管系統的功能。
  6. The authors said stress appears to slow the local, wound - site production of proinflammatory cytokines - - protein molecules produced by white blood cells that play a key role in the early stages of healing

    報告中說,壓力會減慢傷口處"炎癥前期細胞素"的產生速度,這種由白細胞產生的蛋白分子在傷口愈合的早期起至關重要的作用。
  7. The authors said stress appears to slow the local, wound - site production of proinflammatory cytokines - protein molecules produced by white blood cells that play a key role in the early stages of healing

    報告中說,壓力會減慢傷口處「炎癥前期細胞素」的產生速度,這種由白細胞產生的蛋白分子在傷口愈合的早期起至關重要的作用。
  8. " elevated levels of proinflammatory cytokines have been linked to a variety of age - related disease, including cardiovascular disease, osteoporosis, arthritis. certain cancers, and frailty and functional decline, " the study said

    研究表明,炎癥前期細胞素水平增高與各種老年疾病有關,如心血管疾病骨質疏鬆癥關節炎,並有可能引發某些癌癥虛弱和器官功能衰竭等等。
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