traumatic brain injury 中文意思是什麼

traumatic brain injury 解釋
外傷性腦損傷
  • traumatic : adj. 外傷的，損傷的，創傷的；治外傷的。n. 外傷藥。
  • brain : n 1 腦，〈pl 〉腦髓；〈俚語〉計算機；（導彈的）制導系統。2 〈常 pl 〉智力，智能，智慧，腦力；頭腦...
  • injury : n. 1. 損害，毀壞，傷害。2. 傷害的行為。3. 受傷處。
  1. Expression of intercellular adhesion molecule - 1 in human brain cortex after traumatic brain injury and the effect of methylprednisolone

    人腦損傷后皮質細胞間黏附分子1表達及甲潑尼龍的影響
  2. But the relationship among the severity of injury and changes of cytokines are not clear. the mechanisms, post - intervals, severity and prognosis of brain injury are very important in forensic and clinical practice. in this study, brain contusion model of different severity and post - traumatic intervals were made

    Tbi的形成機制、損傷形成時間、損傷程度分析以及預后判斷一直受到法醫及臨床工作者的重視，其中損傷程度的判定成為實際案例中判定案件性質及量刑的主要依據，成為法醫病理學工作者亟待解決的一個重要的科研課題。
  3. Review the theory of diffusion tensor imaging ( dti ) and some of its common clinical application, such as cerebral ischemia, cerebral tumors, brain maturation and traumatic brain injury, also review the potential application of dti in some diseases such as epilepsy, multiple sclerosis

    摘要磁共振彌散張量成像技術是利用水分子的彌散運動各向異性進行成像，是目前唯一反映人體活體組織空間組成信息及病理狀態下各組織成分之間水分子交換功能狀況的檢查方法，可以從細胞及分子水平來研究疾病狀況。
  4. Several genes and cytokines take part in the accommodation of these changes and influence the severity of brain injury, such as tumor necrosis factor a, bel - 2 gene family and ced gene family ( cystein - dependent aspartate - specific protease, caspase ). they are all has relationship with cell death after tbi and control the different cascade of cell death. there are two kinds of cell death after traumatic brain injury ( tbi ) that is necrosis and apoptosis

    腦外傷后腦組織發生的這些改變由許多基因、細胞因子參與調節並影響著損傷程度，除腦水腫外，腦損傷程度主要取決于細胞因子，這些因子包括腫瘤壞死因子- （ tumornecrosisfactor - ， tnf - ） ， b細胞淋巴瘤白血病- 2 （ b - celllymphoma leukemia - 2 ， bcl - 2 ）基因中的bcl - 2 、 bax ， ced基因家族中的半胱天冬酶（ cystein - dependentaspartate - specificprotease ， caspase ）等，它們都與腦損傷后的神經細胞死亡有關，控制著引起細胞死亡的不同層面。
  5. There have been isolated reports in the literature of the natural history and less than complete knowledge of the epidemiology and the pathophysiology of endocrine alteration occurring after traumatic brain injury ( tbi )

    摘要對于腦外傷病人于腦傷后體內內分泌改變的自然病程、流行病學及病理生理學只有少數的報告。
  6. Medical rehabilitation of traumatic brain injury

    創傷性腦損傷的臨床康復
  7. Effects of duxil on cognitive dysfunction in severe traumatic brain injury

    功能訓練結合都可喜對重型顱腦損傷患者認知障礙的康復效果
  8. A study of the neuroprotection effect of 21 aminosteroid u 74389g post traumatic brain injury

    對腦外傷后記憶功能的影響
  9. Traumatic brain injury, tbi

    創傷性腦損傷
  10. Change of nerve cell apoptosis at different time points after traumatic brain injury in rats

    大鼠創傷性腦損傷后不同時間神經細胞凋亡的變化
  11. Pathological, biochemical and molecular biological technique were used to study the changes of bcl - 2, bax, caspase - 1, caspase - 3 and nf - kb after brain contusion aim to investigate the relationship among them and apoptosis and try to provide some objective marker and theory basis to the judgment on post - traumatic intervals and severity of brain injury

    實驗材料與方法實驗材料1 .實驗動物健康wistar大鼠54隻，雌雄不限，體重1809一2809 ，由中國醫科大學實驗動物中心提供。 2 .主要試劑.鼠抗人bel一2 （ e一2 ）單克隆抗體（產品編號: s 。
  12. In the present experiment studies, an acute traumatic model of lateral cortical impact was employed to study expressive changes of microtubule associated protein - 2 ( map - 2 ), cyclooxygenase - 2 ( cox - 2 ), glial cell line - derived neurotrophic factor ( gdnf ), caspase - 3 mrna and protein after brain injury in rats. immunocytochemical staining, western blotting, nucleic acid in situ hybridization with an oligonucleotide probe and computer image analysis were used to detect the dynamic changes of map - 2 mrna, cox - 2 mrna, gdnf mrna, and caspase - 3 mrna in the cortex after moderate traumatic brain injury ( tbi )

    本實驗從自行設計大鼠腦損傷落體打擊器開始，先行建立了一個便於觀察和施加處理因素、控制性好、重復性好的動物模型，選用30g擊錘從25cm高處下落，沖擊應力d為355 . 09kpa ，打擊大鼠右頂部，造成中等程度的閉合性腦損傷，從病理形態學、組織超微結構觀察及微管相關蛋白- 2 （ microtubuleassociatedprotein2 ， map - 2 ） 、環氧合酶- 2 （ cyclooxygenase2 ， cox - 2 ） 、膠質源性神經營養因子（ glialcellline - derivedneutrophicfactor ， gdnf ） 、 caspase - 3基因及蛋白表達的時間性變化，詳盡系統地闡述腦損傷后各指標變化的時間規律性及表達差異可能的形成機制。
  13. Effect of rehabilitation training and nimodipine in severe traumatic brain injury

    康復訓練與尼莫地賓士療重型顱腦損傷
  14. Supplemental magnesium positively affects many of the processes involved in secondary injury after traumatic brain injury and consistently improves outcome in animal models

    動物實驗中，補充鎂可對腦外傷后繼發損傷的許多環節產生積極影響，並持續改善預后。
  15. The study of naloxone on treating severely traumatic brain injury in rat

    納洛酮對重型顱腦損傷大鼠的腦保護機制
  16. Effect of early rehabilitation on patients with severe traumatic brain injury

    重型顱腦損傷患者的早期康復治療
  17. Effect of naloxone on neural functional recovery of rats with traumatic brain injury

    納洛酮對急性顱腦損傷大鼠神經功能恢復的影響
  18. Anatomical integration of newly generated dentate granule neurons following traumatic brain injury in adult rats and its association to cognitive recovery

    外傷性腦損傷后成年老鼠新生齒顆粒狀神經細胞結構整合與認知力恢復的聯系。
  19. Evidence - based treatment for increased intracranial pressure after acute traumatic brain injury

    創傷后顱內高壓患者的循證治療
  20. Clinical study on the rehabilitation of patients with moderate or severe traumatic brain injury

    中重型顱腦損傷患者的康復治療
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