組織酶系統 的英文怎麼說

中文拼音 [zǔzhīxìtǒng]
組織酶系統 英文
tissue enzyme system
  • 組 : Ⅰ名詞1 （由不多的人員組成的單位） group 2 （姓氏） a surname Ⅱ動詞（組織） organize; form Ⅲ量詞（...
  • 織 : 動詞（編織） knit; weave
  • 酶 : 名詞[生物化學] （生物體的細胞產生的有機膠狀物質） enzyme; ferment
  • 系 : 系動詞（打結; 扣） tie; fasten; do up; button up
  • 統 : Ⅰ名詞1 （事物間連續的關系） interconnected system 2 （衣服等的筒狀部分） any tube shaped part of ...
  • 組織 : 1 （組織系統） organization; organized system 2 （組成） organize; form 3 [紡織] weave 4 [醫學] [...
  • 系統 : 1. （按一定關系組成的同類事物） system 2. （有條理的;有系統的） systematic
  1. Methods the diagnosis of chronic gastritis accords with updated sydney classification system, rapid urease test, histopathological staining method and ( superscript 13 ) c breath test were used in detection of hp

    方法慢性胃炎診斷標準參照最新悉尼分類系統，幽門螺桿菌檢測採用快速尿素酶試驗、病理組織學染色及（上標13 ） c呼氣試驗。
  2. Antibodies were used in the macrophage system to inhibit cathepsin d.

    巨噬細胞系統中使用了抗體來抑制組織蛋白酶D。
  3. Antibodies were used in the macrophage system to inhibit cathepsin d

    巨噬細胞系統中使用了抗體來抑制組織蛋白酶d 。
  4. The main study in this paper included as follows : the content and distribution of heavy metals in sediments and benthic organisms from the sewage stream in guangzhou city ; the acute toxicity and joint toxicity of mercury and selenium to swordtail fish ( xiphophorus helleri ) ; the damage of mercury to the indexes of antioxidant system in the gills and livers in swordtail ( including the measurement of the activities of total antioxidative capacity [ t ~ aoc ], superoxide dismutase [ sod ], glutathione peroxidase [ gsh - px ] and the concentration of malondiald - ehyde [ mda ] ) and the relief effects of selenium on it, as well as the physiological damage of mercury on the tissues, namely : the antagonistic effect of na + - k + ~ atpase activity on the tissues between selenite and mercury, and the ultrastructural damage under the exposure of mercury

    研究內容主要有：廣州市河涌沉積物及底棲生物體內重金屬含量及評價；汞和硒對劍尾魚的急性毒性和聯合毒性及安全濃度的評價；汞對劍尾魚鰓和肝臟中抗氧化系統的毒性，包括對總抗氧化能力、超氧化物歧化酶、谷胱甘肽過氧化物酶活力及丙二醛含量的測定及硒對其保護作用；汞對劍尾魚組織生理毒性即：汞對na ~ + - k ~ + atpase活力的影響及硒的保護作用和汞和對劍尾魚組織超微結構的損傷等。以高氯酸?硝酸消化法和火焰原子吸收分光光度法測定了廣州市河涌沉積物和底棲生物中重金屬含量。
  5. Estrogen ( e2 ) is one of the steroid hormones, which is mainly synthesized and secreted by the gonad affecting proliferation, differentiation and function exerting of target tissues. circumferential estrogens were mainly produced by ovary and spermary, and the brain estrogens came mostly from hypothalamus, neurons of limbic system and neuroglias

    雌激素四）是由芳香化酶催化雄激素轉化而來的，它影響靶組織的生長分化和功能發揮，外周雌激素主要由卵巢、睪丸產生，腦內主要由下丘腦、邊緣系統的神經元和神經膠質細胞產生。
  6. Methods : hyperosmotic pressure animal model was established by administering 3 % sodium chloride as drinking water to rats or increasing osmotic pressure of the culture medium. osmoregulation positions in the brain, reciprocal projection pathways between the medullary visceral zone ( mvz ) and supraoptic nucleus ( son ) or hypothalamic paraventricular nucleus ( pvn ), oscillation of intracellular calcium in cultured neurons and astrocytes were studied by means of anti - fos, glial fibrillary acidic protein ( gfap ), tyrosine hydroxylase ( th ) or vasopressin ( vp ) multiple imrnunohistochemical staining, immuno - electronic microscope, wga - hrp retrogradely tracing and cell culture methods. results : ( 1 ) fos positive neurons within the mvz, parabrachial nuclei, locus ceruleus, pvn, son, subfomical organ increased markedly

    方法：通過給予大鼠飲用3氯化鈉或提高培養基滲透壓濃度的方法復制高滲刺激模型，主要採用抗fos 、膠質原纖維酸性蛋白（ gfap ）和酪氨酸羥化酶（ th ） （或加壓素? vp ）免疫組織化學多重染色、免疫電鏡、 wga - hrp束路追蹤結合免疫組織化學多重染色、細胞培養等實驗方法，系統觀察了中樞參與滲透壓反射的調控部位、下丘腦視上核（ son ）神經元? ast超微結構的變化、延髓內臟帶（ mvz ）和son及下丘腦室旁核（ pvn ）之間往返投射通路和神經元的性質及其與ast的關系、培養神經元和ast內鈣波的變化。
  7. The antioxidant abilities of protective enzymes were enhanced in restricted range by hg2 + stress in the early period and the lower concentrations, which were weaken because of the further exposure, especially in liver

    抗氧化酶系統在受到低濃度及染毒初期的一定限度內能被誘導而加強，但隨著重金屬毒害加深而減弱，尤其是肝組織。
  8. Levels of fasting blood glucose and 24h urinary microcontent of albumin 24 h malb were determined dynamically ; the serum glycosyl hemoglobin hba1c was determined after the last medication ; the ultrastructural changes of kidney were observed by transmission electron microscope ; the expressions of collagen, fibronctin, laminin ln, and the ecm metabolism influencing factors, including mmp2, tissue inhibitor of metalloproteinase timp2, transfer growth factor 1 tgf 1 in renal tissue were detected by immunohistological chemistry and image collecting analytical system

    動態檢測各組大鼠空腹血糖fbg 24h尿微量白蛋白24h malb ，末次給藥后測定大鼠血漿糖化血紅蛋白hba1c透射電鏡觀察各組大鼠腎臟超微結構改變，應用免疫組化技術及圖像採集分析系統測定各組大鼠腎臟組織中型膠原c纖維連接蛋白fn層粘連蛋白ln的表達，測定影響ecm代謝的基質金屬蛋白酶2 mmp2基質金屬蛋白酶抑制劑2 timp2及轉化生長因子1 tgf 1的表達。
  9. ( 3 ) pah " levels in maricultural site tvere much higher than that in the adjacent non - maricultural site, and it showed that mariculture was one of the contributors of marine pah " pollution in seal atef. 2 western xiamen harbort ( 1 ) pah " levels in surface seat " ater and sediment of the survey site " ere quite severe

    5系統研究了鱸魚、真鯛體內不同器官、組織的生化指標? ?過氧化氫酶（ cat ） 、超氧化物歧化酶（ sod ） 、脂質過氧化（ lpo ）指標，結合水體及生物樣的pah污染分析結果，探討了這些指標對水環境pah污染的指示作用。
  10. An array of regulatory proteins have been found, which inhibit the formation of central enzymes involved in early stages of the complement activation pathway. these include membrane cofactor protein ( mcp cd46 ), decay - accelerating factor protein ( daf cd5 5 ), complement receptor 1 ( cr1, cd35 ), as well as cd59, which inhibits formation of the membrane attack complex during later stages. these regulatory factors are widely expressed and abundant on many cells, and in fluids of reproductive system

    目前發現，機體多種細胞以及生殖系統的體液中表達和分泌豐富的補體調控蛋白，包括作用於補體活化早期階段的cd55 、 cd46 、 cd35和作用於補體活化終末階段的cd59 ，它們分別通過抑制補體活化過程中關鍵的c3 、 c5轉化酶和抑制形成膜攻擊單位，抵抗補體對自身組織細胞的攻擊。
  11. A male patient of 10 years old gave his chief complaint of slow movement and unclear vision in the xishan department of neural regeneration and functional reconstruction in beijing shijingshan district in october 16th 2005

    0引言x -連鎖腎上腺腦白質營養不良是一種遺傳性代謝疾病，由於飽和極長鏈脂肪酸在過氧化酶體內-氧化障礙，以致飽和極長鏈脂肪酸在血、腦白質、腎上腺皮質等器官和組織大量積聚，引起中樞神經系統脫髓鞘和腎上腺皮質萎縮或發育不良。
  12. This study was to investigate the effects of sulfur dioxide inhalation at different concentrations on some glutathione - related enzymes such as glutathione s - transferase ( gst ), glucose 6 - phosphate dehydrogenase ( g6pd ) and glutathione reductase ( gred ) in brain, lung, heart, liver, kidney and spleen of mice by the technology of biochemical toxicology. the results were showed as follows, so2 exposure at different concentrations caused the changes of glutathione redox system. moreover, the activities of antioxidative enzymes and the contents of reduced glutathione ( gsh ) were decreased significantly in different tissues at higher concentrations of soa

    本研究利用生化毒理學技術研究了不同濃度二氧化硫吸入（ 22 2mg m ~ 3 ， 64 3mg m ~ 3 ， 148 23mg m ~ 3 ）對純系昆明小鼠腦、肺、心、肝、腎、脾六種組織的谷胱甘肽還原酶（ glutathionereductase ， gred ） 、谷胱甘肽硫轉移酶（ glutathiones - transferase ， gst ）和葡萄糖- 6 -磷酸脫氫酶（ glucose6 - phosphmedehydrogenase ， g6pd ）活性的影響，結果表明so _ 2吸入使小鼠不同組織的谷胱甘肽氧化還原系統發生了改變，表現為隨著so _ 2吸入濃度的增加，該系統中的抗氧化酶活性的顯著變化和抗氧化物質水平的顯著降低，且存在著組織差異性。
  13. Methods : in cultured lung explants without serum, the lipid component synthesis of pulmonary surfactant was evaluated in [ 3h ] - choline incorporation ; mrna content of phosphocholine cytidylyltransferase ( cct ) in lung explants was investigated in rt - pcr ; the changes of the ultrastructure of the at ii cells were observed with electron microscope ; the expression of nmdar1 subtype was observed in immunohistochemistry staining ; nitric oxide synthase ( nos ) activity, nitric oxide ( no ) content, superoxide dismutase ( sod ) level, malondialdehyde ( mda ) content and lactae dehydroase ( ldh ) level were determined by biochemistry methods. results : 1. influence of glutamate on synthesis of the lipid component of pulmonary surfactant ? with l - arginine, glu inhibited [ 3h ] - choline incorporation with good dose - dependence and time - dependence ; ( 2 ) mrna content of cct of the glu treatment groups was decreased ; ( 3 ) glu increases the release of ldh in cultured lung explants ; ( dwith electron microscope histochemistry, glu induced the changes of the ultrastruture of at ii iv cells

    方法：採用成年大鼠肺組織無血清培養，運用[ ~ 3h ] -膽堿摻入法測定ps主要脂質磷脂酰膽堿（ pc ）合成量； rt - pcr擴增檢測肺組織中pc合成限速酶磷酸膽堿二胞苷酰基轉移酶（ cct ） mrna含量；透射電子顯微鏡法觀察肺泡型上皮細胞和ps系統超微結構的變化；免疫組織化學染色檢測glu的受體nmdar1亞單位的表達；生化測定肺組織乳酸脫氫酶（ ldh ）釋放量和肺組織勻漿中一氧化氮合酶（ nos ）活性、一氧化氮（ no ）生成量、超氧化物歧化酶（ sod ）水平以及丙二醛（ mda ）含量。
  14. 1. the relationship between protein kinase c and remember behavior alteration of mice exposed to lead. introduction lead has high affinity to nerve and it can be accumulated in nerve tissues, which can cause long - period damage to nervous system, especially to memory and learning function

    一、慢性鉛暴露小鼠腦組織蛋白激酶c活性變化及其與記憶行為改變的關系前言學習記憶是腦的高級功能，其物質基礎是中樞神經系統高度的可塑性，包括神經網路、神經環路及突觸連接等不同水平的可塑性。
  15. Previous studies have showed that sulfonylureas possess a bidirectional effect on cardiovascular system : they can raise the coronary artery tone, aggravate ischemic injury to the cardiovascular tissues, and accelerate cardiac hypertrophy through closing cardiovascular atp - sensitive k ( superscript - ) channels ; meanwhile they can also regulate glucolipid metabolism, modulate inflammatory reaction and inhibit atherosclerosis through activating peroxisome proliferators - activated receptors

    摘要基礎研究表明，磺脲類藥物對心血管系統有雙向影響：磺脲類藥物能夠關閉心血管系統atp敏感性鉀通道，從而增高冠脈張力，加重缺血對心血管組織的損傷和促進心臟肥厚的發生；另一方面，磺脲類藥物能夠激活過氧化物酶體增殖物激活受體，從而調控糖脂代謝，調節炎癥反應和抑制動脈粥樣硬化的發生。
  16. In the present experiment studies, an acute traumatic model of lateral cortical impact was employed to study expressive changes of microtubule associated protein - 2 ( map - 2 ), cyclooxygenase - 2 ( cox - 2 ), glial cell line - derived neurotrophic factor ( gdnf ), caspase - 3 mrna and protein after brain injury in rats. immunocytochemical staining, western blotting, nucleic acid in situ hybridization with an oligonucleotide probe and computer image analysis were used to detect the dynamic changes of map - 2 mrna, cox - 2 mrna, gdnf mrna, and caspase - 3 mrna in the cortex after moderate traumatic brain injury ( tbi )

    本實驗從自行設計大鼠腦損傷落體打擊器開始，先行建立了一個便於觀察和施加處理因素、控制性好、重復性好的動物模型，選用30g擊錘從25cm高處下落，沖擊應力d為355 . 09kpa ，打擊大鼠右頂部，造成中等程度的閉合性腦損傷，從病理形態學、組織超微結構觀察及微管相關蛋白- 2 （ microtubuleassociatedprotein2 ， map - 2 ） 、環氧合酶- 2 （ cyclooxygenase2 ， cox - 2 ） 、膠質源性神經營養因子（ glialcellline - derivedneutrophicfactor ， gdnf ） 、 caspase - 3基因及蛋白表達的時間性變化，詳盡系統地闡述腦損傷后各指標變化的時間規律性及表達差異可能的形成機制。
  17. More intriguingly, in hippocampus, 11 - hsd1 and gr were found to co - exist in the same neuron ; and 11 - hsd1 can convert biologically inert 11 - keto gc metabolites into biologically active gc. gc regeneration by 11 - hsd1 will concentrate gc locally to enable its interaction with the low - affinity gr

    而11 - hsd1具有氧化還原酶的雙重催化作用，廣泛存在於糖皮質激素的靶器官，如肝臟、神經系統、胎盤和胎膜，其催化方向取決于組織類型和功能狀態。
  18. Compared with the quiet state, the rate of sod / mda of red cell, sod / mda x gsh - px / mda of serum were decreased after exercise, it showed : although the activity of antioxidant enzyme was increased, the capacity of antioxidant system was decreased, the tissues or cells might be injured. the concentration of blood locate was increased significantly, which might be one of the reasons causing the exercise fatigue

    2運動后，機體紅細胞sod mda 、血清sod mda 、血清gsh - px mda華唐賦圾大竿洋礦學虜2002厲匝士研究生畢業樁文? ?第二頁共44頁的值同安靜時相比，呈下降趨勢，表明：雖然運動后的抗氧化酶的活性增強，但機體抗氧化系統的能力是下降的，機體組織細胞有可能受到損傷。
  19. The relationship between microbiology and environment changes and human activities influence was assumed based on these results. more than 50 % of the earth ' s surface is covered by deep - sea area

    深海生物經過長期的進化、選擇，在各種極端的環境條件下形成了獨特的組織結構、酶系統及代謝機制以進行生存和繁衍。
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