cell death 中文意思是什麼

cell death 解釋
細胞壞死
  • cell : n 1 小室，單室；隔間，艙；〈詩〉茅舍；（單個的）蜂窩，蜂房。2 〈詩〉墓穴，墓。3 （大修道院附屬的...
  • death : n 1 死，死亡。2 死狀，死法；慘死；死因。3 褫奪公權。4 死刑。5 絕滅，消滅。6 謀殺；慘案。7 〈古語...
  1. Hereby the process of senescence of the antirrhinum majus cotyledon as a embryonic organ appeared some features of programmed cell death ( pcd )

    據此說明子葉在衰老過程中具有某些細胞編程性死亡的結構特點。
  2. Pea ; apical bud ; senescence ; programmed cell death

    豌豆頂芽衰老細胞編程性死亡pcd
  3. Growth factors can act on many kinds of cells as mitotic promoter and deprival of growth factors can cause cease of cell growth or cell death. thus growth factors are usually thought relative to cell growth and differentiation

    生長因子可以作為促分裂劑作用於多種細胞，剝奪生長因子可以導致細胞生長停止或細胞死亡，因此通常認為生長因子與促進細胞的增殖和存活有關。
  4. Cell death is associated with the presence of numerous engorged vacuoles.

    細胞死亡伴隨出現大量充盈的囊泡。
  5. Use pi staining of fixed cells and facs analysis to determine type of u 2 - os cell death induced by bfgf

    採用固定后細胞pi染色流式細胞術檢測bfgf誘導u2 os細胞死亡的形式。
  6. The characteristics of tm - 22 expression presented in transgenic tobacco : 1 ). virus specificity in either homozygote or heterozygote ; 2 ) tm - 22 gene integrated in tobacco genomic dna with single copy and in inheritance and segregation to progenies on the mendel role ; 3 ). transgenic line with tm - 22 promoter ( ptm47 ) showed infected symptoms with cell death distinguished to one with 35s promoter ( ptm49 ) after inoculation with tomv - 2a

    其次，通過氨基酸序列和結構的比較，確定tm - 2 ~ 2基因的編碼蛋白與tomv病毒在抗病反應中相互識別的特異氨基酸及其功能；然後，應用重組dna技術，互換tm - 2 ~ 2基因和tm - 2基因的對應結構域，構建嵌合基因，獲得嵌合蛋白表達的轉化體，驗證tm - 2 ~ 2編碼蛋白中變異氨基酸的作用。
  7. It was also found that the nuclear envelope disappeared earlier at the micropylar end than that at the chalazal end. the speed of the programmed cell death were different in three nonfunctional megaspores at the micropylar end and their relationships with the neighboring cells were also different. the biological significance was discussed about these phenomenon

    還發現了大孢子母細胞合點端的核膜先於珠孔端的消失，並且珠孔端3個大孢子的死亡時間不同，與相鄰細胞的關系也不同，進而討論了這些現象的生物學意義。
  8. Parkinson ' s disease arises from genetic and possibly neurotoxic causes that produce massive cell death of the neuromelanin - containing dopaminergic neurons of the substantia nigra

    帕金森氏病的病因包括基因突變和神經毒性物質作用，導致黑質多巴胺能神經元大量的死亡。
  9. It uses a combination of a photosensitizing agent silicon - pathalocyanine, pc4 and strong visible light. first of all, the photosensitive pc4 is loaded into the cancer cells. when the pc4 is exposed to bright light, it increases the production of no and other oxidative species such as oh and o2 inside the cells, which leads to cell apoptosis disintegration and even cell death

    首先，將光敏感性的pc4注入病人身體內，由於pc4染劑非常容易和不健康的細胞結合，這些不健康的細胞例如癌癥細胞在結合pc4活化性染劑后且又曝露在亮光下時，細胞內一氧化氮no及其它氧化物如氫氧離子oh -或過氧離子o2 -在細胞內的產生量會增加，因而導至此不健康的細胞萎縮，甚至死亡2 。
  10. Itwas observed that sphingomyelin plays anegative regulatory role on the activity of spla 2, resulting in a strong inhibition on spla 2 - induced cell death

    我們觀察到，鞘磷脂對分泌型磷脂酶a的活性起著負2調控作用，它能有效的抑制分泌型磷脂酶a2對細胞的殺傷作用。
  11. Monofunctional alkylating agent n - methyl - n ' - nitro - n - nitrosoguanidine ( mnng ) is a widely spread environmental mutagen and carcinogen that targets dna and proteins to generate adducts. among the adducts, o6 - alkyl guanine is the predominant mutagenic lesion because of its mispairing properties, which can eventually lead to chromosomal aberrations, point mutations, and cell death. this lesion also appears to be involved in tumor initiation, particularly in gastric carcinogenesis

    單功能烷化劑n -甲基- n -硝基- n -亞硝基胍（ mnng ）是一種在環境中廣泛存在的化學誘變劑和致癌劑，它能和dna及蛋白質等生物大分子形成加合物（ adduct ） ，其引起的與突變有關的主要dna損傷類型是o ~ 6 -甲基鳥嘌呤，這種損傷與腫瘤尤其是胃癌的發生密切相關。
  12. However, a - type channel blocker 4 - ap and channel blocker apamin displayed no effect on cell death and percentage of tunel positive cells and caspase - 3 activation

    通道阻斷劑叩amin門卜m ）對缺氧復氧后神經元死亡、 tunel陽性細胞比例以及caspascs激活均無顯著影響，表明bkc 。
  13. " for their discoveries concerning genetic regulation of organ development and programmed cell death

    發現器官發育和細胞程序性細胞死亡細胞程序化凋亡的遺傳調控機理
  14. Brenner shared the nobel medicine prize in 2002 with robert horvitz of the united states and briton john sulston for their work on genetic regulation of organ development and cell death

    布瑞爾與美國科學家羅勃特何維茲和英國科學家約翰薩爾頓三人憑借他們在器官發育和細胞死亡方面的遺傳基因研究成果一舉獲得了2002年的諾貝爾醫學獎。
  15. It is toxic for most mammalian cells since ricin a chain ( rta ) is an rna specific n - glycosidase that removes a specific adenine residue in a highly conservative region from among over 4000 nucloside residues present in 28s rrna, and causes protein synthesis inhibition and cell death

    Rta具有n -糖苷酶活性，可催化28srrna在4234位脫去腺嘌呤，使核糖體60s亞基失活，從而抑制蛋白質合成，導致細胞死亡。 b鏈（ rtb ）是結合鏈，能和細胞表面半乳糖受體結合，協助a鏈進入細胞內。
  16. We first reported al - induced programmed cell death in the root - tip cells of barley

    本研究首次報道了鋁毒誘導大麥根尖細胞程序性死亡。
  17. Labeling tunel method. the cell ultrastructural changes were similar to apoptosis in animal cells : the apical meristemetic cells underwent the programmed cell death. this was first detected in the apex cells of apical meristem, while peripheral cells differentiated gradually into different parts of a floral bud. but all the cells in the floral bud were subjected to the pcd process before it developed into a complete flower. 140bp dna fragment was found to deposit in apical bud during the plant development. the most important role of caspase - 8 was detected by western blot, and the expression of the procaspase - 8 was time - related with the dna frgmentation and the transformation from vegetative to the reproductive growth. these results suggested that pcd was an active process during the differentiation of apical meristem, and the senescence observed in the apical bud was due to the pcd process

    顯微超微結構研究表明，短日照條件下豌豆頂芽的衰老過程是從營養生長錐向花芽的轉化，而用dna原位末端標記tunel caspase - 8 western blot和140 bp dna片斷積累的試驗結果證明，轉化為花芽的整個生長錐細胞發生了編程性死亡pcd ，而且其最頂端部分細胞首先發生pcd ，而頂端周圍的分生組織細胞逐漸分化出花芽的各部分，但頂芽最後並沒有發育成為完整的花，所有細胞就都發生pcd ，從而頂芽衰老。
  18. The results show aocm can enhance the viability of pc 12 cells and reduce zn2 + induced cell death

    結果顯示aocm可提高pc細胞的活性，減輕硫酸鋅誘導的pc細胞死亡。
  19. Spla is able to induce the intracellular ceramide 2 2 generation followed by programmed cell death

    分泌型磷脂酶a能誘導細胞的程序化死亡，並伴隨有細胞2內神經酰胺生成的增加。
  20. Conversely, the intracellular sphingomyelinase ( smase ) can greatly enhance the cell death inducing effects of spla 2

    相反，鞘磷脂酶則能顯著增強分泌型磷脂酶a對細胞的毒性作用。
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