敏化激活 的英文怎麼說

中文拼音 [mǐnhuàjīhuó]
敏化激活 英文
sensitization
  • 敏 : Ⅰ形容詞（靈敏;敏捷） quick; agile; smart; nimble Ⅱ名詞（姓氏） a surname
  • 激 : Ⅰ動詞1 （水因受到阻礙或震蕩而向上涌） swash; surge; dash 2 （冷水突然刺激身體使得病） fall ill fr...
  • 活 : Ⅰ動詞1 （生存; 有生命） live 2 [書面語]（救活） save （the life of a person）:活人無算 （of a goo...
  • 激活 : activation; sensitization; activate; sensitize; active激活材料 active material; 激活劑 activating...
  1. A reliability experiment of thermal aging was carried out for the two types of joints, scanning electron microscopy, energy dispersive x - ray spectrometer and micro x - ray diffractomer were adopted to investigate the interfacial evolution behavior of joints, and kinetics model of imc formation was established. the results show that imc growth follows the parabolic law as a function of aging time at certain aging temperature, imc growth is more sensitive to the aging temperature than the aging time, the activation energy of cu - al imc growth is 97. 1kj / mol and the major forming cu - al imc are cual2 and cu9al4, the activation energy of au - al imc growth is 40. 1kj / mol and the main au - al imc are au4al and au5al2, with au2al and aual at the interfacial periphery of joints, the rate of cu - al imc growth is about 1000 times slower than that of au - al imc, and kirkendall voids and cracks are easily appeared during thermal aging in gold ball bonds while voids and cracks are absent in copper ball bonds even after aging at 200 for 2900 hours and 250 for 169 hours

    研究結果表明:金屬間化合物厚度與老化時間的關系符合拋物線法則，金屬間化合物的生長對老化溫度比老化時間更加敏感; cu - al金屬間化合物生長的激活能為97 . 1kj / mol ，老化后金屬間化合物呈層狀分佈，主要相為cual2和cu9al4 ; au - al金屬間化合物生長的激活能為40 . 1kj / mol ，主要相為au4al和au5al2 ，同時在界面周邊區域生成了au2al和aual ;老化過程中cu - al金屬間化合物生長速率比au - al金屬間化合物生長速率小103數量級;金絲球鍵合點200老化96小時出現了明顯的kirkendall空洞和裂紋，但銅絲球鍵合點200老化2900小時和250老化169小時都沒有形成空洞和裂紋。
  2. Activated agent of cerium that is abundant and cheap together with a little terbiumas additive is used in the study, to research and explore luminescent mechanism of aluminates strontium

    本研究擬選用原料豐富、價格便宜的輕稀土元素鈰作敏化劑，以鋱作激活劑，對其在鋁酸鍶基體中的發光機理這一新領域進行研究和探討。
  3. Cupric nitrate is used as catalyst, oxidizer, activating agent for luminescent powder, and photosensitive resistance materials

    硝酸銅用作催化劑氧化劑熒光粉激活劑及光敏電阻材料等。
  4. Changes of intracellular calcium regulatory signals in pc12 cells during the activation and desensitization induced by anatoxin - a

    12細胞激活和脫敏時胞內鈣調信號的變化
  5. Abstract : plant responses to salt stress via a complex mechanism, including sensing and transducing the stress signal, activating the transcription factors and the corresponding metabolizing genes. since the whole mechanism is still unclear, this review emphasize the biochemical events during the plant adaptation to salt stress referring to an index of importance : the homeostasis in cytoplasm, the biosynthesis of osmolytes and the transport of water. most of these biochemical events were elucidated by study of halophyte and salt - sensitive mutations, also many important genes involved were cloned and used to generate stress - tolerance phenotypes in transgenic plants. on the other hand, about the molecular mechanism in signal transduction, the research of arabidopsis mutations and yeast functional complementation provided helpful traces but not full pathway

    摘要植物對鹽脅迫的耐受反應是個復雜的過程，在分子水平上它包括對外界鹽信號的感應和傳遞，特異轉錄因子的激活和下游控制生理生化應答的效應基因的表達.在生化應答中，本文著重討論負責維持和重建離子平衡的膜轉運蛋白、滲調劑的生物合成和功能及水分控制.這些生理生化應答最終使得液泡中離子濃度升高和滲調劑在胞質中積累.近年來，通過對各種鹽生植物或鹽敏感突變株的研究，闡明了許多鹽應答的離子轉運途徑、水通道和物種特異的滲調劑代謝途徑，克隆了其相關基因並能在轉基因淡水植物中產生耐鹽表型;另一方面，在擬南芥突變體及利用酵母鹽敏感突變株功能互補篩選得到一些編碼信號傳遞蛋白的基因，這些都有助於闡明植物鹽脅迫應答的分子機制。
  6. An mtrp is a hyperirritable spot in a taut band of skeletal muscle fibers with characteristics related to central sensitization in the spinal cord

    肌筋膜激痛點是骨骼肌肉纖維里緊繃肌帶上的過度激活點，伴隨著與脊髓里中樞性敏感化有相關聯的特徵。
  7. Previous studies have showed that sulfonylureas possess a bidirectional effect on cardiovascular system : they can raise the coronary artery tone, aggravate ischemic injury to the cardiovascular tissues, and accelerate cardiac hypertrophy through closing cardiovascular atp - sensitive k ( superscript - ) channels ; meanwhile they can also regulate glucolipid metabolism, modulate inflammatory reaction and inhibit atherosclerosis through activating peroxisome proliferators - activated receptors

    摘要基礎研究表明，磺脲類藥物對心血管系統有雙向影響：磺脲類藥物能夠關閉心血管系統atp敏感性鉀通道，從而增高冠脈張力，加重缺血對心血管組織的損傷和促進心臟肥厚的發生；另一方面，磺脲類藥物能夠激活過氧化物酶體增殖物激活受體，從而調控糖脂代謝，調節炎癥反應和抑制動脈粥樣硬化的發生。
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