肌糖原 的英文怎麼說
中文拼音 [jītángyuán]
肌糖原
英文
muscle glycogen-
The diabetes coronary heart disease high reason of incidence of a disease is many - sided, yet chiefly is the fat supersession chaos, and creates glycerine three grease and the cholesterol to get higher, that sweet and protein are metabolized oddly be able to create myocardium capillary pathological changes and the myocardium supersession chaos, but the flora nervous system chaos easily arouses that blood vessel pulling force adds and injures the artery breastwork
糖尿病冠心病發病率高的原因是多方面的，但主要是脂肪代謝紊亂，造成甘油三脂和膽固醇增高，糖和蛋白質代謝的失常會造成心肌的微血管病變和心肌代謝紊亂，而植物神經系統紊亂易引起血管張力增加而損傷動脈壁。Researches of schistosomiasis vaccines have gone more than 60 years, approximately including from the stages of dead vaccine and live vaccine ( irradiated attenuated cercariae vaccine ) to gene engineered vaccine, etc. many different forms of vaccines have been tested in animal models, including gluthathione s - transferase, paramyosin, irv - 5, triose phosphate isomerase, sm23, fatty acid binding protein ; which were considered promising by who / tdr. but none of them steadily accomplished the pre - set target level of 40 % protection. in order to enhance the protective capacity further, it is essential to develop novel vaccine antigens and / or vaccine adjuvants
血吸蟲病疫苗研究已有60多年的歷史，大致經歷了死疫苗、活疫苗（照射致弱尾蚴疫苗）和基因工程疫苗等研究階段，產生了一些who / tdr推薦認為很有希望的疫苗候選分子，如谷胱甘肽- s -轉移酶（ gst ） 、副肌球蛋白（ sm97 ） 、照射致弱疫苗抗原5 （ irv - 5 ） 、磷酸丙糖異構酶（ tpi ） 、曼氏血吸蟲膜內在蛋白（ sm23 ）和脂肪酸結合蛋白（ fabp ， sm14 ）等，但其對宿主的保護作用均不甚理想，未能穩定地達到40或以上的保護力水平，因此有必要繼續尋找新的疫苗抗原分子和/或疫苗佐劑，進一步提高其保護力。With the conclusion of a burst activity, the lactic acid level is high in the body fluids, leaving the large animal vulnerable to attack until the acid is reconverted, via oxidative metabolism, by the liver into glucose, which is then sent ( in part ) back to the muscles for glycogen resynthesis
由於突發性行為的結果，致使動物體液里的乳酸含量上升，在乳酸通過氧化新陳代謝、在肝臟里形成葡萄糖、回落到正常含量前，動物往往具有較強的攻擊性，那些生成的葡萄糖（一部分）然後輸送回肌肉進行糖原二次合成。Change of the lactic acid when liver is sclerotic occurs an obstacle for liver glycogen process, save up of the lactic acid after muscle activity is overmuch, cause lack of power
肝硬化時乳酸轉變為肝糖原過程發生障礙，肌肉活動后乳酸蓄積過多，引起乏力。Study on the effect of l - arg on inosite content in exhaustive training
精氨酸對肌糖原含量影響的研究Research on the variation principles of the muscle glycogen in rats during repeated bouts of high intensity exercise
重復性大強度運動大鼠在不同時期肌糖原的變化規律Abstract : we have investigated the ultrastructural changes of the biopsied muscle specimens from 8 cases of metabolic myopathies including 3 cases of glycogenosis, 2 cases of lipid storage myopathy and 3 cases of mitochondria myopathy. diagnosis and differential diagnosis with tem for metabolic myopathy as well as the significance of some ultrastructural changes were discussed in this study
文摘:本文對3例糖原累積病， 2例脂質沉積性肌病和3例線粒體肌病共8例代謝性肌病肌活檢標本的超微病理改變進行了觀察分析，對代謝性肌病的超微病理診斷、鑒別診斷以及某些病理改變的診斷意義進行了初步探討。During the initial few days of restricting energy intake, you will lose some water and glycogen ( a type of energy stored in your liver and muscles ) and not much fat
在開始節食的首數天，身體會失去水份和糖原（一種多醣儲存在肝臟及肌肉內，當身體需要時，糖原可以迅速分解為葡萄糖） ，另減去少量脂肪。With powerful cells activating function, the product can activate fiber cells in cuticle, assist to produce proteoglycan, obviously remove wrinkles and firm droopy skin, assist to firm cuticle, improve collagen fiber, make skin firm and rich persistently
能強力賦活真皮纖維細胞，幫助核心蛋白聚糖的生成，顯著改善肌膚皺紋、鬆弛現象，有助於真皮的即效回復，快速補充其彈性，增強膠原纖維，持續保持肌膚緊實而富有彈性，製造出充盈緊致的肌膚。Methods the 54th generation of transformed human embryonic tendon cells and artificial composite materials of carbon fibers ( cf ) and polyglycolic ( pga ) were co - cultured in vitro to construct tet. lt was frozen in liquid nitrogen with four kinds of cpa for 2 months. post - thawed quickly and transplanted into hind limbs of nude mice, and repaired the defects of achilles tendon. after 2, 4, 6, 8, 12 weeks, the morphological, histological, ultrastructure, short tandem repeat loci and immunohistochemistry examination were detected, and biomechanical strength of tet were examined. result tendon cell survived and could secret type i collagen after 12 weeks to transplanted into nude mice. in the group of dmso + raffmose + kh2o4, vacuole in mitochondrion degraded i tendon cell ranged in order, abundant collagen fibers were found and linked each other and the biomechanical strength was increased as time elapsed. c onclusion dmso + raffmose + kh2o4 could protect tet in deep low temperature
組織工程肌腱制備完成後在四種抗凍劑保護下液氮凍存2月；快速復溫后植入裸鼠以修復跟腱缺損， 2 、 4 、 6 、 8 、 12周后取出，觀察形態學、組織學、電鏡和免疫組織化學變化，短串聯重復位點檢測和生物力學變化。結果實驗組組織工程肌腱體內植入12周后仍有肌腱細胞存活並分泌型膠原；隨著時間延長， 10二甲基亞碸（ dmso ） +棉子糖（ 30mmol l ） + kh _ 2po _ 4 （ 25mmol l ）組線粒體空泡減少，肌腱細胞排列整齊，膠原纖維增粗並連接，抗拉強度增高。But where glucose forms tight bonds with the long - lived protein collagen, the result is a constellation of changes, including thickened arteries, stiff joints, feeble muscles and failing organs - the hallmarks of a frail old age
然而，人體中的萄葡糖一旦與生命期長的膠原蛋白結合就會產生一系列重大變化，諸如動脈變厚，關節僵硬，肌肉乏力，器官衰退等年邁體虛的特微。The new study, published july 16, 2007, in the proceedings of the national academy of sciences ( pnas ), demonstrates that insulin resistance in skeletal muscle ? caused by decreased ability of muscle to make glycogen, the stored form of carbohydrate from food energy ? can promote an elevated pattern of lipids or fats in the bloodstream that underpins the metabolic syndrome
2007年7月16日，發表在美國國家科學院學報（ pnas ）的最新研究結果顯示：骨骼肌發生胰島素抵抗時，肌肉製造糖原（從食物中獲得的碳水化合物的儲存形式）的能力減弱；這將會使脂質和脂肪在血液中含量增加，並因此引起代謝綜合征。分享友人