足黴素 的英文怎麼說
中文拼音 [zúméisù]
足黴素
英文
podomycin-
Based on the residue monitoring results in the previous year, especially on the feedback information of the positive ( non - compliance ) results, and taking account of world - wide alert notifications, samples of some items to be taken will be duly increased year by year ; based on the reasonable suggestions in fvo inspection report and the problems of chloramphenicol and nitrofuran which had been notified by eu in recent years, the monitoring items are being added in order to meet the residue requirement of importing countries and regions such as the eu, japan, korea, switzerland and hk
國家質檢總局根據上一年度殘留監控結果反饋情況，特別是陽性結果的檢出情況，結合各國預警通報的情況，每年適當增加對應監控的檢測項目的取樣數量；尤其是針對歐盟每次考察報告中對我殘留監控計劃提出的合理化建議和近年來歐盟通報的氯黴素和硝基呋喃問題，增加了滿足歐盟、日本、韓國、瑞士、香港等國家和地區殘留監控要求的項目。The resulting plasmid, named prok - sod2, was mobilized to agrobacterium tumefaciens strain gv3101 used for plant transformation. the yeast sod2 gene was introduced into arabidopsis thaliana ( ecotype landsberg erecta ) by agrobaterium tumefaciens - mediated transformation with floral - dipping method under the control of camv 35s promoter. transformants were selected for their ability to grow on medium containing kanamycin ( 30mg / l ), several homozygous lines that were all tolerant to kanamycin were selected and used for further molecular and physiological determination
本實驗將sod2基因構建到植物表達載體prok中，導入農桿菌后，進行植物遺傳轉化，實現其在擬南芥中過量表達，在含30mg l的卡那黴素的培養基上篩選獲得純合轉基因株系，自交一代獲得足夠的純和轉基因種子后，對其進行了分子生物學的驗證及生理指標的檢驗。The mechanism of chloramphenicol toxicity is apparently failure of the newborn to conjugate chloramphenicol with glucuronic acid due to inadequate activity of hepatic glucuronyl transferase
氯黴素中毒的機制顯然是由於新生兒肝臟的葡萄糖醛酸轉移酶活性不足，不能使氯黴素與葡萄糖醛酸結合。This ， in combination with inadequaterenal excretion of drugin the newborn, results in a higher than expected level of chloramphenicol in the plasma
這（一機制） ，加之新生兒腎臟的藥物排泄不足，結果導致在血漿中的氯黴素水平升高。The mechanism ofchloramphenicol toxicityis apparently failure of the newborn to conjugate chloramphenicol withglucuronic aciddue toinadequate activityof hepaticglucuronyl transferase
氯黴素中毒的機制顯然是由於新生兒肝臟的葡萄糖醛酸轉移酶活性不足，不能使氯黴素與葡萄糖醛酸結合。分享友人