胰抑素 的英文怎麼說

中文拼音 [yíyìsù]
胰抑素 英文
pancreastatin
  • 胰 : 名詞[生理學] （胰腺） pancreas
  • 抑 : Ⅰ動詞（向下按; 壓制） restrain; repress; curb Ⅱ連詞[書面語]1 （表示抉擇） or 2 （表示轉折） but3 ...
  • 素 : Ⅰ形容詞1 （本色; 白色） white 2 （顏色單純） plain; simple; quiet 3 （本來的; 原有的） native Ⅱ名...
  1. Bitter melon can clear stomach heat and boost eyesight. perhaps the best substantiated use today is that of bitter melon for people with diabetes mellitus. other properties and actions documented by traditional use are antifungal, antiparasitic, antivenin, bitter, cardiotonic tones, balances, strengthens the heart, digestive stimulant, emetic causes vomiting, menstrual stimulator, purgative strong laxative, vermifuge expels worms

    苦瓜含豐富的維他命c葉酸鉀及維他命b雜，另有鉻chromium及一種類似胰島素的植物蛋白質，能增強胰島素功能，有降血糖和有助促進血液中糖份進入細胞的功效，加快能量使用，預防糖尿病，可以幫助血糖的控制，抑制脂肪的分解還有獨特的蛋白質，具有抗病毒的作用等等。
  2. Have again, fat person have high cholesterol mostly hematic disease and tall insulin hematic disease, after the cholesterol inside body is elevatory, the cholesterol content of the cellular film such as cell of lymphocyte, huge bite is elevatory, controlled immune function

    再有，肥胖者大都有高膽固醇血癥和高胰島素血癥，體內膽固醇升高后，淋巴細胞、巨噬細胞等細胞膜的膽固醇含量升高，抑制了免疫功能。
  3. Thus, it is postulated that in the presence of mild neutropenia, whose function is inhibited by effect of insulin excess, the bacillus was able to find a port of entry, probably via micro - abrasions of the bowel mucosal lining

    因此，推測可能存在中性粒細胞減少，功能被過多的胰島素抑制，芽孢桿菌可以乘虛而入，很有可能通過腸內壁粘膜微破損進入循環。
  4. The objective of this study is to evaluate the effects of active immunization against ss on nutrient digestion and metabolism, growth and carcass composition of the growing hu - sheep ; 2, on the ss secretion in fundus, antrum and pancreas and the ablity of the antibodies to integrate free ss ; 3, on passage rates of digesta ; 4, on activities of digestive enzymes in pancreas and digesta ; 4, on gene expression of gh in pituitary

    本項研究內容集中於後者，主要的試驗目的是： 1生長抑素主動免疫對ss進行免疫中和對生長期湖羊生長，營養物的消化，代謝，胴體組成影響； 2對胃竇膜，基底膜和胰腺ss分泌以及ss抗體結合ss的能力影響； 3
  5. The endocrine cells in the digestive and glands of alligator sinensis embryos aged from 8th to 55th day were localized and compared by using immunohistochemical method with thirteen kinds of antiseras of hormone. during the development of pancreas in alligator sinensis embryos, somatostatin ( ss ) immunoreactive ( ir ) cells, 5 - hydroxytryptamine ( 5 - ht ) - ir cells, glucagon ( glu ) - ir cells, epidermal growth factor ( egf ) - ir cells appeared on 18th day. no p53 protein - ir cell, gastrin - ir cell, testosterone - ir cell, chromogranin a - ir cell, vasoactive intestinal polypeptide - ir cell, epithelial membrane antigen - ir cell or insulin - ir cell was found in the pancreas of alligator sinensis embryos

    本實驗採用免疫組織化學技術，應用13種不同的抗血清，對孵育時間8 ? 55天揚子鱷胚胎消化道及消化腺內分泌細胞的種類進行鑒別、定位和比較，結果如下：揚子鱷胚胎胰腺中，生長抑素、 5 ?羥色胺、胰高血糖素、表皮生長因子、胰多肽免疫反應陽性細胞出現于第8天； p物質免疫陽性細胞出現于第18天； p53 、胃泌素、睪酮、嗜鉻素a 、血管活性腸肽、上皮膜骯原、胰島素免疫陽性細胞在各期揚子鱷胚胎胰腺中均未發現。
  6. Continue metformin. additionally continue sulfonylureas when starting basal insulin therapy. - glucosidase inhibitors may also be continued

    繼續應用二甲雙胍。開始應用基礎胰島素治療時還可以繼續應用磺脲類藥物。 -糖苷酶抑制劑也可以繼續使用。
  7. May also inhibit the hyperfunctions of islet a cells notably, thereby impel them to be atrophic and disappear, so as to reduce the volume of glucagon produced. such a miraculous change promotes the balancing of pancreas functions and hence returns the blood sugar to normal

    同時亦發現胰島修復因子「降糖素- deona 」又具顯著地抑制胰島a細胞過亢的功能，從而促其萎縮消失，以減少胰高血糖素的分泌量。
  8. In addition, the stomach contained glu - ir cellsn gas - ir cells and vip - ir cells, and the duodenum contained glu - ir cells and gas - ir cells. different endocrine cells varied from one to another in the distribution pattern, the stage of appearance and the number. no glu - ir cell, gas - ir cell or vip - ir cell was found in the oesophagus and no vip - ir cell in the duodenum

    另外，胚胎期揚子鱷消化道內分泌細胞的種類和分佈與成體有差異，胚胎期消化道檢出胰高血糖素、胰多肽和p物質免疫陽性細胞，而成體未檢出；胚胎期消化道中胃泌素、血管活性腸肽，生長抑素免疫陽性細胞分佈與成體消化道不同。
  9. And afp - ir cells appeared in the outer membrane of liver or blood vessel endothelial neared the outer membrane only on 55th day. ss, 5 - ht, glu, pp, sp, egf and afp were not expressed in all liver cells

    各期肝細胞均未發現表達胰高血糖素、胰多肽、生長抑素、 5 -羥色胺、 p物質、表皮生長因子、甲胎蛋白。
  10. This article begins with a brief overview of the discovery of glucagon and the contributions that somatostatin and a sensitive and selective assay for pancreatic ( vs. gut ) glucagon made to understanding the physiological and pathophysiological roles of glucagon

    文章的開始部分簡單回顧了胰高糖素的發現過程，以及其對生長抑素的作用和一項具有高敏感性和特異性的檢驗胰腺中的胰高糖素的方法（與腸道內的檢測法相比） ，來幫助人們理解胰高糖素的生理和病理生理作用。
  11. " these findings have potential clinical implications, as they suggest that long - term rosiglitazone usage in the treatment of type 2 diabetes and insulin resistance may cause osteoporosis, owing to a combination of decreased bone formation and increased bone resorption, " the authors conclude

    作者總結認為，上述研究發現對于臨床治療具有潛在的意義， 2型糖尿病及胰島素耐受患者長期使用羅格列酮治療，可能通過抑制骨生成與促進骨的再吸收而導致骨質疏鬆
  12. Combining the ace inhibitor and the arb, " which target different aspects of the renin angiotensin system, may hae merit in the treatment of both cardioascular and metabolic disease, which are characterized by reciprocal relationships between endothelial dysfunction and insulin resistance, " the inestigators conclude

    Ace抑制劑聯合arb ， 「針對腎素血管緊張素系統的不同方面，在治療內皮功能障礙與胰島素抵抗相互關聯的心血管和代謝病方面有優勢， 」研究人員得出結論。
  13. Such therapies may also provide additional advantages in islet transplantation, as the authors of the publication speculate that the immunosuppressive therapies could also contribute to the loss of islet function over time that led to loss of insulin independence in several of the patients

    耐受治療還可能提供胰島移植的附加優勢，正如作者推測的，隨著時間延長，免疫抑制治療會損害胰島功能，使一些患者的胰島素非依賴性消失。
  14. The key mechanisms involved are inhibition of protein - phosphotyrosine phosphatases and activation of nonreceptor protein - tyrosine kinases, in an insulin - receptor tyrosine kinase independent fashion

    釩在生物體內發揮作用的主要機制是通過蛋白磷酸酪氨酸磷酸酯酶的抑制和非受體蛋白酪氨酸的激活一種與胰島素受體完全不同的途徑發揮作用的。
  15. By inducing 3t3 - l1 preadipocyte with individual hormone, it was identified that the mitotic clonal expansion was induced only by insulin, not by 1 - isobutyl - 3 - methylxanthine or dexamethasone

    有報道用藥物抑制了克隆擴增的同時抑制了細胞的分化。我們發現克隆擴增是僅由胰島素誘導引起的。
  16. In summary, we used the cdna microarrays to analyze the differentially expressed genes in the arsenic trioxide induced k562 cells. our results further confirmed in gene expression level that growth arrest and apoptosis could be induced by arsenic trioxide. such induced apoptosis may also involve the functional inhibition of igf

    應用基因晶元技術研究aszo3作用前後k562細胞的基因表達變化，與以往的研究結果相一致，也是通過抑制細胞生長、誘導細胞凋亡而發揮抗腫瘤的作用，在這個過程中， aszo3可能對胰島素樣生長因子有抑制作用。
  17. The insulin after the meal is secreted rise, can restrain decompose adiposely, the origin of energy is restricted

    4 .飯后胰島素分泌上升，可抑制脂肪的分解，能量的來源就受到限制。
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